Renal hypertension is mainly due to the elevation of blood pressure caused by renal parenchymal lesions and renal artery lesions, which is called renal hypertension in symptomatic hypertension. The pathogenesis and pathological features: First, the pathological features of renal parenchymal disease are manifested by glomerular vitreous degeneration, interstitial tissue and connective tissue hyperplasia, tubular atrophy, and narrowing of the small renal arteries, resulting in both substantial damage and inadequate blood supply to the kidney. Second, the proliferation of mucinous muscle fibers in the middle layer of the renal artery wall forms most of the small aneurysms, causing the inner wall of the small renal arteries to protrude in a bead-like pattern, resulting in segmental narrowing of the renal arteries. Third, non-specific aortitis, causing inadequate perfusion of renal blood. What tests should be done for renal hypertension? 1.Ultrasound examination: If one side of the kidney is found to be shrunken, it may be a narrowing of the renal artery on this side; renal artery Doppler ultrasonography can understand whether there is narrowing of the main trunk of the renal artery, but obese people often cannot see clearly because of the intestinal organs and the left renal artery. 2.Nuclide renal artery scan: the development of about 20 seconds, radioactive reagents with blood flow filling the abdominal aorta, renal artery and perfusion of the renal vascular bed, you can observe the blood flow of the renal vessels. 3.Renal arteriography: It is the golden indicator, which can show whether the lesion is unilateral or bilateral, the site and scope of the lesion, the presence or absence of collateral circulation and the renal parenchymal visualization. However, the cost is higher, and it also requires arterial cannulation, which has certain risks. 4.Digital subtraction: It only requires the injection of contrast agent and is a non-invasive test. However, when creatinine is elevated, both of these methods cannot be used to avoid acute tubular necrosis caused by contrast agent and aggravate renal function damage.
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