Inflammation caused by bacterial invasion of the endometrium after the destruction of the physiological defenses of the female reproductive system in general, in the presence of low body resistance, is called endometritis. It is divided into two types: acute endometritis and chronic endometritis. Acute endometritis mainly occurs after childbirth, miscarriage or official surgical operation, especially informal abortion, with pathogenic bacteria such as Escherichia coli, Staphylococcus and Streptococcus. There is pain in the lower abdomen, a feeling of falling, and a large amount of purulent discharge from the vagina. If the disease is not controlled in time, the inflammation may continue to spread upstream and develop into acute tubal inflammation, ovarian inflammation or pelvic inflammatory disease. If the treatment is not complete, the condition may change from time to time, prolonging over time and forming chronic endometritis. Simple acute nonspecific endometritis usually has little effect on fertility after healing, but in a few cases, endometrial adhesions can form and cause infertility. Since acute endometritis can sometimes cause tubal and ovarian inflammation, these inflammations can be a chronic process that does not dissipate for a long time, thus seriously affecting fertility. Tuberculous endometritis, on the other hand, can seriously affect fertility and is one of the major causes of female infertility. When abortion is performed several times, it affects the thickness of the endometrium, often resulting in an endometrium that is too thin for pregnancy. In fact, the normal thickness of endometrium is 0.8 – 1.2 MM. If the endometrium is too thin, the fertilized egg will have no place to lay and it will be difficult to get pregnant, while if it is too thick, it will lead to irregular menstruation and affect ovulation, thus leading to infertility. The dangers of endometritis are as follows: Firstly, endometritis may affect the number of sexual encounters due to severe pain or may lead to ovarian malfunction or blockage of the fallopian tubes, which may affect fertility. Second: After sperm enter the uterine cavity, inflammatory factors such as bacterial toxins and leukocyte phagocytosis cause sperm death or reduced motility, which reduces the number of sperm entering the fallopian tubes and thus affects fertility. Again: After fertilization, the fertilized egg does not easily implant in the inflamed endometrium, or the antiendometrium body causes obstruction to implantation, resulting in infertility. Unstable fertilization of the fertilized egg makes it extremely easy to miscarry, leading to infertility. Finally, endometritis can also affect the fetus, causing malformations, miscarriage, premature birth, premature rupture of membranes, neonatal infections and later growth disorders.