Cirrhosis is a chronic disease characterized by decompensated liver function, liver fibrosis and pseudolobular tissue formation, and can be clinically divided into two stages: compensated stage and decompensated stage, and the general term early cirrhosis refers to compensated stage cirrhosis. The compensated stage of cirrhosis generally has no obvious symptoms, sometimes there may be sleepiness and weakness, loss of appetite and indigestion, or even diarrhea, but generally not characteristic. This can usually be relieved by rest or other adjustments, which can easily lead to misdiagnosis and eventual progression to the decompensated stage. Symptoms of the decompensated stage of cirrhosis include both hepatic decompensation and portal hypertension. Decompensated liver function is manifested by indigestion, aversion to greasy food, malnutrition, and in severe cases, jaundice begins to appear. In addition, prothrombin time is prolonged due to impaired synthesis of coagulation factors. Also, hypersplenism due to splenomegaly may present with signs of hematocrit and hemorrhage. The activation and inactivation of hormones also need to pass through the liver, so some symptoms of endocrine disorders may appear, such as abnormal sex hormones, menstrual irregularities and infertility in women, and decreased libido, hair loss, breast development, spider nevus, and liver palms in men. The manifestations of portal hypertension are usually detected by examination, and are commonly found in the formation of portal vein collateral circulation, esophagogastric fundic varices, abdominal wall varices, hemorrhoidal varices, retroperitoneal anastomotic branch varices, splenorenal shunt, etc. Long-term portal hypertension can also lead to hypersplenism, splenomegaly, and in more severe cases, peritoneal effusion. Continued development of cirrhosis in the decompensated stage can also lead to a number of complications. For example, vomiting of blood and tarry stools, usually caused by bleeding from ruptured esophagogastric fundic varices. In addition, the accumulation of fluid in the peritoneal cavity can lead to peritonitis or, in more severe cases, hepatic encephalopathy due to the accumulation of toxic substances in the blood. In addition, it can lead to hepatorenal syndrome, hepatopulmonary syndrome, and cancer. Therefore, cirrhosis is a disease with no obvious symptoms in the early stage, but with extensive damage in the later stage. It is difficult to rely on symptoms to diagnose in the early clinical stage, and regular medical checkups are needed for timely examination.