The mechanism of cerebral palsy treatment is mainly accomplished by blocking the spinal reflex r loop. As we all know, the vertebral system is composed of two levels of neurons, the vertebral cells located in the precentral gyrus and the anterior cortex of the central paracentral lobule are the upper motor neurons; the lower motor neurons are the brain nerve somatic motor roots and the anterior horn motor cells of the spinal cord located in the brainstem. Normal reflex activity in the upper has an inhibitory effect on the lower motor neurons, and damage to either upper or lower motor neurons will cause skeletal muscle paralysis. Cerebral palsy is defined as damage to upper motor neurons. Loss of inhibition of lower motor neurons results in increased excitability of lower motor neurons, increased muscle tone, hyperactive tendon reflexes, and muscle spasm without atrophy. It is caused by overstimulation of the pull reflex through the spinal cord. Its receptor is the muscle shuttle, which is innervated by r-motor neurons, and the afferent fibers of the muscle shuttle consist of class I fibers. The efferent fibers that innervate skeletal muscle consist of two types of a and r motor neurons. a regulates the extra-saccadic muscle; r regulates the intra-saccadic muscle and sensitizes the receptors. SPR is to cut off the afferent fast-transmitting fibers as much as possible, block the r-loop in the spinal reflex, reduce the excitability of proprioception, selectively retain some slow-transmitting fibers with high excitability threshold, and preserve the limb sensory function, so as to release the spasm and restore the ability of random movement and self-control. At the same time, reducing the excitability of the cortical center improves spasticity.