The incidence of tuberculosis is still high in Qinghai due to the backward economy and the poor health care awareness of the people, and the incidence of constrictive pericarditis due to tuberculosis is high. The youngest patient admitted to our hospital was 9 years old, and we now present a briefer discussion on the treatment of constrictive pericarditis. Chronic constrictive pericarditis is a disease in which chronic inflammation of the pericardium leads to thickening, adhesions, or even calcification of the pericardium, which limits the diastole and contraction of the heart, decreases cardiac function, and causes systemic blood circulation disorders. Chronic constrictive pericarditis is mostly caused by tuberculous pericarditis. The generally thickened pericardium binds the heart and bruises all organs of the body, showing signs such as jugular vein anger, hepatomegaly, ascites, and pleural fluid. Tuberculous pericarditis may present with symptoms 3 to 6 months after the acute phase. Commonly, fatigue, shortness of breath, dysuria, abdominal distension, loss of appetite, ascites, hepatomegaly, and increased dyspnea in those with generalized edema. Electrocardiogram, echocardiogram, X-ray examination can be diagnosed. Once the diagnosis of chronic constrictive pericarditis is established, surgery should be performed as soon as possible. Preoperative preparations should be made according to the patient’s condition. The patient should be prepared according to the patient’s condition, such as sodium restriction, appropriate application of diuretics (tachyphylaxis, dihydrocoumarol), maintenance of water-electrolyte balance, strengthening nutrition, protein, vitamin, small amount of blood or plasma transfusion, anti-TB treatment for tuberculosis patients, and appropriate amount of elimination of pleural fluid ascites, etc. Signs: jugular venous anger, hepatomegaly, ascites, swelling of the lower extremities, and increased heart rate. The diagnosis of typical constrictive pericarditis is not difficult based on clinical manifestations and laboratory tests. Clinical differentiation from cirrhosis, congestive heart failure, and tuberculous peritonitis is often required. The clinical manifestations and hemodynamic changes of restrictive cardiomyopathy are very similar to this disease, and it may be very difficult to distinguish between the two, and endomyocardial biopsy is required for diagnosis if necessary. Treatment options:Once the diagnosis is confirmed, early consideration should be given to pericardial debridement surgery after the acute symptoms have subsided to avoid myocardial atrophy and compromise the efficacy of the surgery. Bed rest should be given before surgery. A low-salt diet and diuretics should be given as appropriate. Those with anemia and reduced serum protein should be given supportive therapy to improve their general condition, and those with active tuberculosis should be actively treated with anti-tuberculosis therapy before and after surgery. For those with longer duration of disease and more pronounced cardiac decompensation, cardiac stimulants, such as small doses of cetiran or digoxin, can be given before or after surgery to prevent heart failure in the atrophied myocardium after increasing the burden. Those with pericardial calcification alone without increased venous pressure do not need special treatment. Those with poor myocardial response to cardiac stimulants or very poor liver and kidney function should not undergo surgery. Pericardial dissection is an effective treatment for constrictive pericarditis, and 90% of those who survive after surgery have significant improvement in symptoms and return to the workforce. Therefore, early surgery is currently advocated, that is, surgery can be performed when the pericardial infection is basically controlled clinically. Patients who are operated too late often have atrophy and fibrous degeneration of the myocardium, and although the surgery is successful, there is little improvement in the postoperative situation due to myocardial lesions, and even heart failure occurs because the degenerated myocardium cannot adapt to the increase in blood flow into the heart. If treatment of constrictive pericarditis is delayed, most patients are disabled or die due to failure, ascites and peripheral edema or serious cardiac complications. If thorough pericardial debulking surgery is performed early, most patients can achieve satisfactory results.