Traumatic wet lung is a comprehensive lesion of lung tissue congestion, interstitial edema or hemorrhage caused by chest injury. In severe thoracic trauma, the thoracic cavity suddenly shrinks, the pressure in the thoracic cavity suddenly increases, the terminal small bronchi and alveolar capillaries rupture to form edema and hemorrhage, and after the external force is removed, the thorax returns to its original position, generating instant negative pressure in the thoracic cavity and further aggravating the original injury area. After severe trauma to the chest, a large amount of exudate retention in the terminal small bronchi and alveoli and interstitial lung, bronchial blockage occurs, which further increases the airway resistance, causing ventilation dysfunction, resulting in hypoxia, increasing capillary permeability of lung tissue, more fluid entering the lungs, and alveolar gas exchange becomes impaired, resulting in ventilation dysfunction. In addition, severe chest wall pain, destruction of thoracic stability, chest muscle spasm, paradoxical breathing and other factors aggravate the body hypoxia, at this time, the partial pressure of pulmonary venous oxygen decreases, the partial pressure of carbon dioxide increases, appearing dyspnea, cyanosis, hemoptysis or pink foam sputum, irritability, tachycardia, and even blood pressure drops. For patients with severe traumatic wet lung, various effective treatment measures should be actively taken: 1. Oxygenation. It can increase blood oxygen concentration, reduce pulmonary edema and bronchospasm, improve ventilation and gas exchange, and improve hypoxemia. 2.Keep the airway open. Encourage patients to cough up and excrete sputum, and for those who cannot effectively excrete sputum, nasal cannula or fiberoptic bronchoscope can be used to aspirate sputum, and tracheal intubation or tracheotomy when the effect is poor is more conducive to clearing tracheal and bronchial secretions. 3.Adequate and effective analgesia. Analgesia is beneficial to breathing and coughing and sputum excretion. Pain makes patients inhibit breathing and coughing, resulting in inadequate ventilation, increased dead space, accumulation of secretions, further aggravating pulmonary edema, followed by the development of hypercapnia, hypoxemia, resulting in respiratory distress, especially when accompanied by chronic obstructive pulmonary disease, can rapidly develop into ARDS, oral analgesics or lidocaine and prednisolone intercostal nerve block anesthesia to relieve pain. 4, rib fracture to perform chest wall fixation, multiple multiple rib fractures can produce paradoxical breathing and mediastinal oscillation due to chest wall softening, resulting in respiratory and circulatory impairment, and its mortality rate can reach 43%-50%. Therefore, effective fixation of the floating chest wall, as soon as possible to correct the paradoxical breathing, release the intrathoracic pressure, keep the airway unobstructed, actively maintain respiratory function, as soon as possible to reopen the lung, is an important rescue and treatment measures, with patients with hemopneumothorax to perform closed chest drainage or dissection to stop bleeding, repair the bronchial fissure. 5, control the infusion rate and the input of crystalloid fluid, and appropriately increase the input of colloid fluid to avoid aggravating pulmonary edema and accelerate the absorption of pulmonary edema. After chest crush injury or blunt force injury, there can be organism stress reaction, microcirculatory disorder, bronchospasm, and increased alveolar permeability, while the alveolar wall is damaged and blood plasma enters the alveoli, causing hypoxia and changes in pulmonary capillary permeability and increased fluid in the lungs. 6, respiratory insufficiency, especially for severe traumatic wet lung with hypoxia and hyperCO2emia, artificial ventilator assisted breathing should be carried out, commonly used end-expiratory positive pressure ventilation 5-10cmH2O, which can effectively improve alveolar and interstitial edema, promote the reopening of the non-dilated lung, improve lung compliance, ensure adequate oxygen supply and gas exchange, and at the same time can overcome chest wall softening, have a fixed effect on the floating chest wall However, positive end-expiratory pressure ventilation may aggravate or produce pneumothorax and affect cardiac reflux and expulsion, so it should start from a low value and be adjusted while observing. In this group of patients, 10 cases used ventilator with satisfactory results. 7.Rational use of hormones and diuretics. 8.Actively treat combined injuries. 9.Prevent or control infection with antibiotics, traumatic wet lung has susceptibility to infection, so all should be given broad-spectrum antibiotics at an early stage. 10, early prophylaxis with drugs that inhibit stomach acid, in order to reduce the occurrence of stress ulcers. In conclusion, traumatic wet lung is a common and serious chest trauma, mainly early diagnosis, active improvement of respiration, reduce pulmonary edema, anti-infection, reasonable application of hormones and diuretics, timely treatment of combined injuries, if necessary, the use of ventilator-assisted breathing and other comprehensive treatment measures, treatment effect is good, the cure rate is high.