Seborrheic alopecia is generally referred to as androgenetic alopecia, a non-scarring type of baldness that occurs during and after puberty, and is mainly characterized by follicular miniaturization and progressive hair loss due to elevated androgens.
There is a genetic predisposition to the disease, and androgens are one of the important mechanisms in the pathogenesis of the disease. Androgenetic alopecia hair loss area scalp follicle type II 5α-reductase activity is significantly higher than the non-alopecia area, and the 5α-reductase enzyme in the tissues can convert testosterone to 5α-dihydrotestosterone (DHT).
DHT and the androgen receptor on the hair follicle cells combined to play a biological role, making the hair follicle miniaturization, anagen hair gradually thinning, hair growth cycle is shortened, so that the original coarse black hair gradually become light-colored cui-ui hair, and ultimately, due to the disappearance of hair follicle shrinkage, the cui-ui hair loss, the formation of the forehead, the coronal area to the top of the head baldness.
Seborrheic alopecia patients should seek timely medical treatment, standardized treatment under the guidance of the doctor.