Whether a patient with electrolyte disorder can heal on its own depends on the patient’s condition, the type and severity of the electrolyte disorder. In most cases, patients with mild symptoms and no dehydration can gradually heal on their own without special treatment and without excessive worry. The daily electrolyte intake and output of the normal body is relatively constant and regulated by a variety of factors, the main ions include sodium, potassium, calcium, phosphorus and magnesium. However, if electrolyte disorders occur, you should go to the hospital to ask professionals to determine the type and severity of electrolyte disorders, exclude some emergencies, and treat accordingly. If it is asymptomatic or mild hyponatremia, usually no treatment is needed to deal with the primary disease and restrict water intake on the basis of high salt and high protein diet, patients can usually heal themselves. In acute and symptomatic hyponatremia, especially in the presence of severe neurological symptoms, such as headache, drowsiness, altered consciousness, epilepsy, immediate treatment with diuretics and arginine pressor receptor antagonists is required; 2. Hypernatremia: common in patients with low water intake, profuse sweating and burns, if only physiological water intake is insufficient, mild patients can usually be self-healing. However, if there is an underlying disease, the primary disease should be actively treated, and after early replenishment of sufficient water, then appropriate oral rehydration salt to replenish electrolytes. Some patients with mild or asymptomatic diseases can be self-healed by adjusting their diet and replenishing potassium. Severe hypokalemia often requires intravenous potassium supplementation under close supervision. 2. Hyperkalemia: It is common in patients with hemolysis, massive burns, and abnormal renal potassium excretion. Since hyperkalemia can affect neuromuscular as well as cardiac functions, and in severe cases, respiratory muscle paralysis and cardiac arrhythmias can occur and endanger life, so emergency treatment is required, and patients are given intravenous calcium and insulin to reduce blood potassium concentration. Third, disorders of calcium metabolism: 1. Hypercalcemia: Patients can develop fatigue, drowsiness, and hypotonia due to decreased muscle excitability, which is usually difficult to heal on its own. The primary disease should be actively treated, supplemented with saline, and usually treated with glucocorticoids; 2. Hypocalcemia: If it is caused by a low-calcium diet, the intake of calcium-containing foods can be enhanced, and some patients with mild cases can heal on their own. In severe cases with convulsions and muscle spasms, intravenous calcium supplementation should be administered, and vitamin D can also be used in combination. iv. disorders of phosphorus metabolism: patients’ symptoms are usually atypical or masked by the accompanying symptoms of other electrolytes, often diagnosed by laboratory tests, and usually not self-cured. Patients with mild cases usually do not require special phosphorus supplementation or phosphorus excretion treatment, and symptomatic treatment of the primary disease is sufficient. Severe hypophosphatemia patients need to take oral milk or phosphorus preparations, while severe hyperphosphatemia can be drained through drugs or dialysis phosphorus. Fifth, magnesium metabolism disorder: usually hypomagnesemia patients, can appear neuromuscular and cardiac muscle excitability increased, manifested as muscle tremors, ataxia and frequency disorders. So if the symptoms of patients, often need to take oral magnesium enzyme preparations, and asymptomatic patients can temporarily do not supplement magnesium, may be self-healing. If hypomagnesemia is present, some mild patients can be self-cured by stopping magnesium-containing drugs, but severe patients can develop drowsiness, bradycardia or even life-threatening, and need intravenous calcium gluconate to antagonize myocardial toxicity.