With the improvement of people’s living standard, high fat, high sugar and other food intake, resulting in excess nutrition, excessive accumulation of fat in the body and overweight and obesity, fatty liver can not be ignored as a factor, but not the only cause. The current prevalence of weight loss, excessive hunger can also cause liver metabolic disorders, resulting in a large accumulation of fat in the liver, can also “starve” out fatty liver. Long-term hunger or gastrointestinal tract digestion and absorption disorders, resulting in a lack of protein in the body, a lack of raw materials for the formation of apolipoproteins, triglycerides in the liver accumulation and fatty liver. Excessive alcohol consumption can “drink” fatty liver, a large number of long-term alcohol consumption: ethanol can cause hepatocyte metabolic disorders, so that lipid acid synthesis increased, oxidation decreased, the blood lipid acid content increased. In addition, the drinker’s appetite is much reduced, and the intake of choline in food is reduced, which inevitably makes the liver lipoprotein synthesis obstructed, and leads to excess triglycerides difficult to remove, resulting in fatty liver. In addition, certain drugs and chemicals, such as overdose or close contact with tetracycline, arsenic, silver, mercury, trichlorethylene, carbon tetrachloride, yellow phosphorus, barbiturates, aflatoxin, etc., can block the synthesis of apolipoproteins, and triglycerides in the liver cannot be metabolized and excreted, thus causing fatty liver accumulation in the liver. According to statistics, about 50% of diabetic patients can develop fatty liver, and about 25% of fatty liver patients have diabetes. When suffering from inflammatory infections, tuberculosis, bacterial pneumonia and sepsis, the integrity of the hepatocyte membrane is destroyed, resulting in abnormal fat metabolism in the liver or hepatocyte hypoxia and fatty liver. Others: Cushing’s syndrome, hyperthyroidism, anterior pituitary hyperfunction, chronic ulcerative colitis, Crohn’s disease, ulcer disease, chronic hepatitis and pregnancy can affect fat metabolism and lead to fatty liver. The clinical manifestations of fatty liver are diverse, and mild fatty liver has no clinical symptoms and is easily overlooked. According to statistics, more than 25% of fatty liver patients can be clinically asymptomatic, and some only have a feeling of fatigue, so at present, fatty liver patients are mostly found by chance during physical examination. Mild enlargement of the liver may be painful to the touch, slightly tough, with blunt edges and smooth surface, and a few patients may have splenomegaly and liver palms. When there is excessive fat deposition in the liver, it can cause severe pain or pressure pain in the right upper abdomen due to swelling of the liver peritoneum and pulling of the liver ligament. What are the dangers of fatty liver? Fatty liver patients are often accompanied by hyperlipidemia, which increases blood viscosity and can induce or aggravate hypertension, coronary heart disease, which can easily lead to sudden death due to myocardial infarction, and can also lead to encephalopathy fatty liver syndrome, also known as visceral steatosis encephalopathy. The mechanism is still unclear, and the death rate is as high as 70-80%. In severe cases of fatty liver, cirrhosis, liver failure and liver cancer can occur. The end result of various liver diseases is often cirrhosis, and fatty liver is no exception, with a higher chance of cirrhosis secondary to hepatocellular. Acute gestational fatty liver disease has a high mortality rate. The clinical manifestations are often similar to those of acute severe liver, which can lead to acute liver failure, pancreatitis, renal failure, systemic coagulation abnormalities and rapid death, mostly in pregnant women in their first pregnancy. Fatty liver can induce or aggravate diabetes mellitus. Diabetes mellitus and fatty liver are a pair of difficult brothers and sisters, and having both will make the treatment more difficult and accelerate the development of the disease. Hepatitis B combined with fatty liver accelerates the progression to cirrhosis. The liver is the largest reticuloendothelial cell phagocytic system. Fatty degeneration or necrosis of liver cells reduces the immunity of the liver. The stomach, intestines, liver and gallbladder are all important organs of the digestive system. The body’s intake of the three major nutrients needs to be metabolized by the liver in order to be used by the body. The impaired liver function of fatty liver patients will involve the spleen, gallbladder, stomach and intestines over time, causing damage to the body’s digestive system.