Adhesive Intestinal Obstruction (AIO) is a condition in which intestinal contents cannot pass smoothly in the intestine due to intestinal adhesions in the abdominal cavity caused by various reasons. When the passage of intestinal contents is blocked, a series of symptoms such as abdominal distension, abdominal pain, nausea and vomiting, and bowel obstruction can be produced. It belongs to the category of mechanical intestinal obstruction, which can be divided into acute intestinal obstruction and chronic intestinal obstruction according to the urgency of onset; complete intestinal obstruction and incomplete intestinal obstruction according to the degree of obstruction; high small bowel obstruction, low small bowel obstruction and colonic obstruction according to the site of obstruction; simple intestinal obstruction and strangulated intestinal obstruction according to the blood supply to the intestine. The disease can be partially treated by non-surgical treatment, but most of the recurrent attacks or conservative treatment is ineffective and still need to receive surgical treatment. Strangulated intestinal obstruction is mostly acquired, except for a few congenital factors in the abdominal cavity, such as congenital developmental abnormalities or meconium peritonitis. The common causes are abdominal inflammation, injury, bleeding, and abdominal foreign body, mostly seen after abdominal surgery or abdominal inflammation, among which adhesions after abdominal surgery are currently the first cause of intestinal obstruction, in addition to abdominal radiotherapy and abdominal chemotherapy can also lead to adhesive intestinal obstruction. Pelvic surgery (e.g., after gynecological surgery, appendectomy, and colorectal surgery) and lower abdominal surgery are particularly prone to intestinal adhesions and intestinal obstruction because the pelvic small bowel is more free, whereas the upper abdominal small bowel is relatively fixed. However, intestinal obstruction does not always occur in patients with intestinal adhesions, and the occurrence of adhesive intestinal obstruction does not necessarily mean that there are extensive and severe abdominal adhesions. Adhesive intestinal obstruction can only occur when the intestinal tube forms an acute angle at the point of adhesion, when the intestinal collaterals are bound by the fixed ends of the adhesive bands, or when a group of intestinal collaterals are adherent to a group and the intestinal wall is scarred and narrowed. The main clinical manifestations of adhesive intestinal obstruction are the symptoms of mechanical intestinal obstruction: abdominal pain, vomiting, abdominal distension, and cessation of defecation. (1) abdominal pain: in intestinal obstruction, there are often paroxysmal abdominal colic due to enhanced intestinal peristalsis. During the onset of abdominal pain, patients often feel that there is gas running in the abdomen, they can see or feel the intestinal pattern and hear high-pitched intestinal sounds; if it is incomplete intestinal obstruction, when the gas passes through the obstruction, the pain suddenly reduces or disappears; if the mesentery is excessively stretched, the pain is persistent and increases paroxysmally; in the late stage of the disease, due to the excessive dilatation and weak contraction of the intestinal tube above the obstruction, the degree and frequency of pain are reduced; when intestinal paralysis occurs, the abdominal pain changes to persistent abdominal distension. (2) Vomiting: The frequency of vomiting, the amount of vomiting and the nature of vomit varies with the location of the obstruction. In high small bowel obstruction, vomiting appears earlier and more frequently, and the amount of vomit is more; in low small bowel obstruction and colonic obstruction, vomiting appears later and less frequently, the amount of vomit is less, and the vomit often has a fecal odor. (3) Abdominal distension: abdominal distension is caused by dilatation of the intestinal canal during obstruction. The degree of abdominal distension varies depending on whether the obstruction is complete and the site of the obstruction. The more complete the obstruction, the lower the site, the more obvious the abdominal distension; sometimes, although the obstruction is complete, but due to the loss of intestinal storage function, vomiting early and frequently, abdominal distension may not appear; if this situation is not noted, it may lead to missed diagnosis and misdiagnosis. If the obstructed intestine forms closed collaterals, it often shows asymmetric abdominal distension, and sometimes dilated intestines can be found there. (4) Stop defecation: because of the obstruction of intestinal content transport, the anal canal stops defecation. However, it must be noted that the intestinal contents distal to the obstruction site can still be sent down by peristalsis. Therefore, even if the obstruction is complete, the patient can continue to have bowel movements until the contents are cleared, and only after they are cleared does the patient stop having bowel movements. Of course, in the case of incomplete obstruction, the defecation does not disappear completely. In addition, the clinical symptoms of intestinal obstruction include disturbance of water, electrolyte and acid-base balance, and in case of strangulated obstruction and intestinal necrosis, shock, peritonitis and gastrointestinal bleeding may occur.