The cervical disc degeneration consists of a nucleus pulposus, a fibrous ring, and upper and lower cartilage plates that form a complete anatomical unit. The cervical intervertebral disc maintains the intervertebral height, absorbs shock, conducts axial compression forces, and maintains stress balance in all directions of cervical spine activity, a function that is accomplished entirely by the interaction of the various structures that make up the disc. Therefore, degeneration of the intervertebral disc is the main factor in the occurrence and development of cervical spondylosis. 1, the nucleus pulposus: is rich in water, has good elasticity of mucin, white, containing chondrocytes and fibroblasts, water content of more than 80% when young. As age increases, the water content capacity decreases, and can be less than 70% in old age. The amount of water content in the intervertebral disc determines its intrinsic level of pressure regulation and elasticity. The normal state of the intervertebral disc accounts for 20% to 24% of the total length of the cervical spine, and its height decreases year by year due to the decrease in water-holding capacity. Early water loss and water absorption decreases, resulting in a corresponding decrease in the volume of the nucleus pulposus, and its normal tissue structure is gradually replaced by fibrous tissue. In cases of increased local stress, trauma and strain, the degeneration can be made more rapid and the pressure inside the disc increases. The degeneration of the nucleus pulposus and the degeneration of the fibrous annulus make the elastic modulus of various parts of the disc change, and the nucleus pulposus may protrude to the edge through the fissure of the fibrous annulus. The nucleus pulposus is most likely to protrude posteriorly due to the weakness of the posterior longitudinal ligament and the low modulus of tension of the corresponding fibrous ring. The degenerated and sclerotic nucleus pulposus may also pass through the posterior longitudinal ligament fissure and enter the spinal canal, directly producing clinical symptoms. Fibular ring: After about 20 years of age, the fibular ring begins to degenerate. The early stage is the transparent degeneration of fibrous tissue, fiber rent and disorderly arrangement, and then cracks. In addition, most of the occupations nowadays are often used to the flexed neck position, so that the nucleus pulposus is squeezed to the posterior side, so the fibrous ring fracture is more common on the posterior side. The early degenerative stage of the fibrous ring has the potential to abort its development if the causative factors can be eliminated early. If once the fissure is formed, it is difficult to recover due to the lack of good local blood supply. 3, cartilage plate: the degeneration of cartilage plate is mainly manifested as the degeneration of function. Studies have shown that the cartilage plate is equivalent to the central zone of the nucleus pulposus site has a semi-permeable membrane role. This role is closely related to the water content performance and nutrient metabolism of the nucleus pulposus. When the fibrocartilage degeneration, even the fibrous ring and the nucleus pulposus lose nourishment, intensifying its degeneration. 4, degeneration of the disc edge: degeneration of the disc edge includes early soft compression of the ligament-disc gap and late fibrosis, calcification or ossification as well as changes in the tissue in front and on both sides of the disc. (1) Formation of the disc-ligament complex: due to degeneration of the pushed disc, the nucleus pulposus tissue prolapses posteriorly under high pressure up to the underside of the ligament, resulting in increased local pressure, which separates the posterior longitudinal ligament from the posterior edge of the vertebral body and forms a prismatic gap. (2) The formation of vertebral body posterior edge bone superfluous: the formation of vertebral body posterior edge bone superfluous is firstly due to the instability of vertebral joint after disc degeneration. After the instability of the vertebral joint, the vertebral body becomes compensatory hypertrophy, mainly manifested as osteophytes at the stress collection point of the anterior and posterior edges of the vertebral body. Osteomalacia formed as a result of multiple stress changes over time is often hard in texture. The formation of osteophytes can also be caused by the granulation tissue in the ligament-disc space that is continuously enlarged and hardened by mechanization, ossification or calcification under the stimulation of repeated trauma and strain.