The main pathological changes in stress urinary incontinence are endogenous urethral sphincter weakness and excessive downward movement of the posterior angle of the bladder urethra, with the latter being more common and a few patients having both. According to the relationship between urinary storage and voiding, the physiological functions of a normal person can only be accomplished when the bladder forceps and urethral sphincter muscles complement each other and work in close synergy. Problems with either of the two groups of muscles will result in urinary dysfunction. And women are more prone to urinary incontinence than men for many reasons, the common reasons are: 1, women are born with a shorter urinary tract, easy to urinary tract infection; 2, pregnancy and birth pelvic muscle ligament relaxation, poor rest after birth, premature weight bearing; 3, more children, or poor repair of birth injuries, resulting in pelvic floor muscle fascia defects; 4, after entering menopause, the function of the ovaries to synthesize estrogen significantly reduced, the urethra and bladder The mucous membrane of the neck atrophies and the wrinkled wall disappears, resulting in a weakening of the power to close the urethra. Young women who have had both ovaries removed due to disease can also suffer from stress incontinence due to low estrogen levels; 5. Direct or indirect injury to the bladder and urethral tissues after pelvic surgery, etc.