Do you know the danger of hyperthyroidism? Hyperthyroidism is a condition in which the thyroid gland synthesizes and releases too much thyroid hormone, causing hyper-metabolism and sympathetic excitation, resulting in palpitations, sweating, increased eating and stool intake, and weight loss. Most patients also often have symptoms such as proptosis, eyelid edema, and loss of vision. Most patients also have symptoms such as protruding eyes, eyelid edema, and loss of vision, as well as varying degrees of goiter, hand tremors, and vascular murmurs in the neck. Hyperthyroidism is particularly prevalent in women, and white-collar and menopausal women are particularly susceptible to hyperthyroidism. Hyperthyroidism can cause complications such as fantasy, mania, schizophrenia and depression. However, hyperthyroidism can cause excessive production of thyroid hormones, which can lead to tachycardia, arrhythmia, menstrual disorders, amenorrhea and infertility in women, and even after pregnancy, it can lead to fetal failure, miscarriage, stunting and stillbirth. Even after pregnancy, the fetus is prone to miscarriage, maldevelopment and stillbirth. Hyperthyroidism can lead to tachycardia, arrhythmia (such as premature beats and atrial fibrillation), increased blood pressure, heart enlargement, heart failure, angina pectoris, myocardial infarction, and other hyperthyroid heart diseases. The longer the duration of hyperthyroidism, the higher the possibility of hyperthyroid heart disease, and it takes longer for hyperthyroid heart disease to recover after hyperthyroidism is cured, and individual patients cannot even return to normal. Atrial fibrillation can also produce blood clots, which, when dislodged, can lead to cerebral embolism and paralysis. Patients with hyperthyroidism also develop hypermetabolic syndrome. Patients are usually hot and sweaty, and hyperthyroidism is often characterized by low fever, but severe patients may have high fever, marked hyperphagia, but weight loss and fatigue. Hyperthyroidism causes liver damage due to increased oxygen consumption, resulting in insufficient oxygen supply to the liver, increased catabolism, increased depletion of liver glycogen, and excessive consumption of various nutrients such as amino acids and vitamins.