The spinal cord is located in the spinal canal, and as people grow and develop, the spinal canal grows faster than the spinal cord, so the lower end of the spinal cord rises gradually in relation to the lower end of the spinal canal. Spinal cord tethering is a condition in which the lower end of the spinal cord cannot rise normally due to various reasons, making its position lower than normal. It is one of the main pathological mechanisms of various congenital developmental abnormalities leading to neurological symptoms, resulting in a series of clinical manifestations known as spinal cord tethering syndrome, also known as spinal cord tethering syndrome. Etiology Various congenital developmental abnormalities of the spinal cord and spinal ends can lead to spinal cord tethering, such as occult spina bifida, spondylolisthesis, spinal cord spondylolisthesis, spinal cord end filament tension, lumbosacral intraspinal lipoma, congenital cysts, and latent hair sinus. In addition to the aforementioned congenital factors, postoperative adhesions in the lumbosacral spinal bulge can also lead to spinal cord reembolization. It is generally believed that spinal cord embolism causes blood circulation disorders at the end of the spinal cord, resulting in the corresponding neurological symptoms. Clinical manifestations The clinical manifestations of spinal cord embolism syndrome (spinal cord tethering syndrome) are similar to those of spina bifida, mainly in the following aspects: 1. Skin changes in the lumbosacral region. Bulging or depressed lumbosacral skin, possibly with discharge or infection; hirsutism; bulging large masses ……. These predict the presence of occult spina bifida, latent hair sinus, spinal bulge, etc., which may be combined with spinal cord embolism. 2. Motor disorders of the lower extremities. The manifestations are abnormal walking, weakness, deformation and pain of the lower limbs, and may be combined with scoliosis. 3.Sensory disorders of the lower limbs. It manifests as abnormal sensation and pain in the lower limbs, perineum and low back. 4.Dysfunction of urination and defecation. Common manifestations include urinary retention, difficulty in urination, urinary incontinence, more frequent urination, less than normal volume each time, etc.; constipation, constipation, or incontinence. Diagnosis and examination Magnetic resonance imaging (MRI) is the main method to diagnose spinal cord embolism, which can not only clarify the presence of spinal cord embolism, but also other coexisting pathological changes, such as lipoma, spinal cord longitudinal bifida, etc. X-ray plain film can determine the presence of spina bifida. Ultrasound and urodynamics of the urinary tract are performed to evaluate the extent of urinary tract involvement and spinal cord neurological impairment in conjunction with urinary and fecal function. Treatment Surgical release of the embolus and removal of lesions such as lipomas.