Spinal cavernous disease is a chronic, progressive lesion of the spinal cord characterized by the formation of tubular cavities in the spinal cord (mainly gray matter) and glial (non-neural cell) hyperplasia, often in the cervical spinal cord. The cause of spinal cavernous disease is unknown. The age of onset is usually 20 to 30 years, with more women than men. Clinical manifestations are: 1. slow progression of the disease: the earliest symptoms appear in a segmental distribution, first involving the upper extremities, and when the cavity gradually expands it can involve the long conduction tracts in the white matter of the spinal cord and develop long tract dysfunction, which can be separated by several years; 2. sensory disturbances: dissociative sensory disturbances manifested by segmental distribution of pain and temperature loss, intact or mildly diminished tactile and deep sensation; 3. Motor disorders: damage to lower motor neurons after involvement of the anterior horn of the spinal cord, manifested by atrophy and weakness of small muscles of the hand (such as interosseous muscles and interphalangeal muscles) and ulnar muscles of the forearm with tremor of the muscle bundles, gradually spreading to the scapular girdle and some intercostal muscles, hypotonic or absent tendon reflexes, hypotonia; cone bundle signs below the cavity, increased muscle tone and hyperactive tendon reflexes, disappearance of abdominal wall reflexes and raphe reflexes, and appearance of pathological reflexes. If the cavity is in the lumbosacral region, the above-mentioned motor and sensory disorders of the lower limbs may appear; 4. common symptoms, and radicular pain or tenderness to touch is also more common.