Spinal cavernous disease is a slowly progressive degenerative lesion of the spinal cord in which the central canal of the spinal cord enlarges or forms a canalicular cavity under the influence of causative agents, often surrounded by glial proliferation. The age of onset is usually 20-30 years, occasionally occurring in childhood or later in adulthood. The disease progresses slowly, with the earliest symptoms often appearing in a phasic distribution, affecting the upper extremities first. As the cavity gradually enlarges, the long tracts of the spinal cord are also involved due to pressure or gliosis, and conduction tract dysfunction occurs below the level of the cavity. The main clinical manifestations are: 1. Sensory abnormalities: if the cavity is located in the cervical and upper thoracic segments of the spinal cord, there will be unilateral segmental sensory disturbances in the upper extremities and upper thoracic segments, such as unilateral pain and temperature disturbances, and if the lesion invades the anterior union, there may be bilateral pain and temperature loss in the hands, ulnar side of the arms or part of the neck and chest, while tactile and deep sensation is intact or relatively normal, called dissociative sensory disturbances. Patients are often found to have pain and warmth deficits only after burns or stabbing or cutting injuries have occurred. 2, motor disorders: cervicothoracic spinal cord cavity appears flaccid partial paralysis of one or both upper limbs, manifested as muscle weakness, decreased muscle tone, especially the small muscles of the hands, interosseous muscle atrophy is the most obvious, severe cases will be claw-shaped hand deformity, and there may be “meat jump” (muscle bundle tremor). Upper motor neuron partial paralysis and increased muscle tone occur in one or both lower limbs. 3, autonomic damage symptoms: the cavity involves the sympathetic spinal center of the lateral horn of the spinal cord, Horner syndrome (cervical sympathetic nerve paralysis syndrome), the corresponding segment of the limb and trunk skin less sweaty, lower temperature, fingertips, nails hyperkeratosis, atrophy, loss of luster. Due to the loss of the generalized temperature sensation, burns and injuries are likely to occur. The loss of pain sensation in the joints can cause joint wear, atrophy and deformity. In advanced stages, patients may develop urinary and fecal disorders. The onset of the disease in adulthood, the presence of segmental distribution of dissociative sensory disorders in other congenital defects, and muscle atrophy of the hands and upper extremities are all features of the disease, and its further diagnosis relies on appropriate CT and MRI examinations.