Segmentalhyalinizing vasculitis, also known as atrophieblanche and livedoidvasculitis, is still controversial. It is characterized clinically by purpura and necrosis on the lower legs and ankles, leaving ivory-white atrophic spots after healing. Cyanotic vasculitis is a vasculitis disease in which polymorphic skin damage occurs on the basis of small vascular lesions in both lower extremities, leaving atrophic scarring after healing. It mostly occurs in young and middle-aged women. The majority of cases are aggravated in summer and reduced in winter, and the opposite is rarely true. Note that cyanotic vasculitis is distinguished from reticulocutaneous cyanosis. Intradermal injection of corticosteroids such as trimethoprim, etc. Low molecular dextrose, anticoagulants, ganglion blockers, niacin, sulfapyridine, etc. are effective for active lesions and can prevent recurrence. Stanozolol (Conradron) and Danazol can also be tried to increase fibrinolysis. Dipyridamole, enterosoluble aspirin, vitamin E and the Chinese herbal medicines Radix Rehmanniae and Salviae are also effective in cyanotic vasculitis. The pathological manifestation of cyanotic vasculitis is the deposition of fibrin-like material in the wall and lumen of superficial dermal capillaries, the formation of thrombi, and necrosis of the vessel wall. Therefore, the treatment is mainly anticoagulation: the main drugs are aspirin, dipyridamole, low molecular heparin, and dextran. In recent years, Danazol has been found to be more effective for cyanotic vasculitis, and its mechanism is to both inhibit coagulation and promote fibrinolysis. Usually 200mg daily is given for 1-2 weeks to significantly improve symptoms.