Tuberculous pleurisy is an inflammatory disease caused by Mycobacterium tuberculosis infection. Its main clinical manifestations are fever, cough, chest pain, dyspnea and pleural fluid accumulation. When Mycobacterium tuberculosis enters the pleural cavity and the body is in a highly allergic state, it may cause exudative pleurisy. I. Diagnosis: The onset resembles a cold, with fever, cough, chest pain, dyspnea and shortness of breath when there is more fluid accumulation, and some have night sweats, fatigue, and loss of appetite. Physical examination reveals that: dry pleurisy has restricted respiratory movement on the affected side, localized pressure pain, palpable pleural friction, and pleural friction sounds on auscultation. In exudative pleurisy with more pleural fluid, the affected side is full, the rib space is widened, respiratory motion and fibrillation are weakened, the trachea and heart are shifted to the healthy side, the turbidity of the clasp below the fluid level, the breath sounds are weakened or disappeared, and the bronchoalveolar breath sounds can be found above the fluid level, and occasionally there are small alveolar sounds. In the case of right pleural effusion, the hepatic turbid tone boundary disappears. If there is pleural adhesion hypertrophy localized thoracic subsidence, restricted respiratory motion buckling turbid sound, breath sounds weakened. On blood examination, blood leukocytes may be slightly elevated in the early stage, and blood sedimentation is increased rapidly, greater than 20 mm per hour in women and 15 mm per hour in men. Pleural fluid examination is consistent with tuberculous exudate, and positive results may be obtained for tuberculosis bacilli. On ultrasound exploration, a liquid flat segment is seen on the affected side of exudative pleurisy. x-ray examination of dry pleurisy may be unremarkable, and images of pleural effusion are seen in exudative pleurisy. Second, the treatment: tuberculous pleurisy to drug therapy, to carry out regular anti-tuberculosis therapy for regular x-ray examination. If exudative pleurisy has a high volume of chest water, compression of the heart and lungs, breathing difficulties, panic, chest tightness and other symptoms, you must immediately go to the hospital for treatment. Third, prevention: In prevention, first of all, we must cut off the transmission route and minimize contact with patients with tuberculosis. Life and living should be regular, appropriate arrangements for work and rest, do not be too tired. Diet should be rich in nutrition, avoid eating spicy, fire-producing phlegm products, and those who have a taste for alcohol and tobacco should resolutely give up. Usually, you should keep your mind relaxed and happy, and participate in some recreational activities that you like. Fourth, the general problem: 1, why is the chest pain more powerful after pumping chest water? Such patients often have more pleural fluid when they visit the clinic, which completely separates the two layers of pleura (pleura is divided into dirty layer and wall layer pleura, and the sensory nerves are mainly in the wall layer), so the friction between the two layers of pleura is reduced so the pain is less, when the pleural fluid is pumped, the pleural fluid in the pleural cavity is reduced, and the friction between the two layers of pleura is increased so the pain is obvious, but if the water continues to decrease, the pain will also slowly disappear. 2.Why should I take hormone? Pleurisy is a highly metamorphic reaction of the pleura to the stimulation of tuberculosis bacteria and its metabolites, and its pathological changes are manifested as congestion, exudation and edema. Glucocorticoids are used for the treatment of tuberculosis mainly because of their non-specific anti-inflammatory and anti-toxic effects, which can reduce capillary dilation in the lesion area, decrease the permeability of the blood vessel wall and cell membrane, reduce exudation and inflammatory cell infiltration, stabilize the intracellular lysosomal membrane, protect the linear stereo, improve the antiviral ability of human cells, and play anti-inflammatory, anti-allergic and antiviral roles, thus reducing the inflammatory response of the pleura and relieving clinical symptoms.