Frontal lobe epilepsy is a more complex type of epilepsy that originates in the frontal lobe and is characterized by simple partial, complex partial, and mixed seizures secondary to generalized seizures or the presence of these seizures, accounting for about 20%-30% of all types of partial seizures. Frontal lobe seizures can sometimes be confused with psychogenic pseudoseizures and can often be combined with status epilepticus. Frontal lobe seizures are usually short in duration; 2. Complex partial seizures originating in the frontal lobe are often accompanied by mild postictal confusion or absence of consciousness; 3. They can soon cause secondary generalized seizures, which are more common in frontal lobe epilepsy than in temporal lobe epilepsy; 4. Tonic postural or hyperkinetic automatism is a prominent feature; 5. Discharges that occur on both sides are often accompanied by falls. Because frontal lobe seizures can affect more adolescents and children, they are of greater concern to patients and parents. The frontal cortex is a relatively large brain area that includes most of the regional structures, and depending on the area affected by the discharge, different forms of seizures can occur. The most common form of seizure is the supplementary motor area (SMA) seizure, which is the most classic form of seizure, with postural focal tonicity with vocalizations, speech pauses, and fencing-like postures. The patient’s head and eyes are turned to the opposite side of the epileptic origin, and the upper extremity on the opposite side of the epileptogenic focus is abducted, the shoulder is externally rotated, and the elbow is flexed, as if the patient is looking at his or her hand. The upper and lower extremities on the ipsilateral side of the epileptogenic focus are tonicly abducted, with more pronounced movements of the distal upper extremity than the lower extremity. This ipsilateral extension of the upper extremity to the side of origin and flexion of the contralateral upper extremity has been described as a “fencing-like posture”. In the case of cingulate gyrus seizures, the form is usually complex partial with motor gestures and autonomic symptoms, such as altered mood and emotion, are common. In the case of prefrontal polar area seizures, the form includes compulsive thinking or onset of contact loss and head-eye turning movements, which may be accompanied by reversal of movement and axial clonic jerks with falls and autonomic symptoms. Orbitofrontal seizures, which mostly take the form of a complex partial seizure with onset motor and gestural automatisms, olfactory hallucinations and delusions, and autonomic symptoms. Dorsolateral frontal seizures, which may manifest as tonic or rare clonus with head-eye rotation and cessation of speech. Insular seizures include mainly symptoms of mastication, salivation, swallowing and larynx, with speech arrest, epigastric aura fear, and autonomic symptoms. Simple partial seizures, especially partial clonic facial seizures, are common and may be unilateral, and if secondary sensory changes occur, numbness may be a symptom, especially in the hand region, and taste hallucinations are also common in this region. Motor cortical seizures are mainly characterized as simple partial seizures, with different sides and different sites of involvement and clinical manifestations. The EEG of epilepsy originating in the frontal lobe is very diverse and complex and requires routine special recording electrodes and long-range EEG monitoring. Autosomal dominant nocturnal frontal lobe epilepsy (ADFNLE) is a recently identified idiopathic localized epilepsy that is currently receiving widespread attention. This type of frontal lobe epilepsy mostly develops before the age of 20, with a male to female ratio of 2:1, and is an autosomal dominant type with incomplete episodes. Sudden head lifting, head shaking, head tilting back, upper limb lifting or throwing-like movements, lower limb hyperextension, circling, bicycle-like movements or rhythmic movements of the limbs. Some patients are aware of the seizure but cannot control it, can hear external sounds but cannot respond, and can recall the seizure afterwards. Consciousness is restored immediately after the seizure, and rarely there is post-ictal disturbance of consciousness or post-ictal headache. The above symptoms suggest that the seizure originates from or involves the frontal supplementary motor area. 80% of medications are effective and 30% can be completely controlled. Nocturnal frontal lobe epilepsy with autosomal dominant inheritance has characteristic clinical seizure features and a low rate of positive EEG changes during seizures and interictal periods, which should be distinguished clinically. Frontal lobe epilepsy is prone to seizures at night, so sleep EEG has important diagnostic value for epilepsy during sleep seizures, especially idiopathic nocturnal frontal lobe epilepsy. In conclusion, frontal lobe seizures are generally exaggerated, and some patients do not have loss of consciousness, and the seizures are short-lived and occur in clusters at night. The symptoms of the child should be effectively controlled.