Intermittent claudication refers to the patient from the beginning of walking, or after walking a certain distance (usually about a few hundred meters), unilateral or bilateral lumbago, numbness and weakness of the lower limbs, and even limp, but a little squatting or sitting down after a few moments of rest, the symptoms can quickly alleviate or disappear, the patient can still continue to walk, and then after walking for a period of time, the above symptoms reappear. Because the limp occurs intermittently in this process, it is called intermittent claudication. The appearance of neurogenic intermittent claudication is mainly due to the pathological basis of the existing stenosis of the lumbar spinal canal, the increased pressure load on the vertebral body and nerve roots during uprightness, plus the stretching and contraction activities of the lower limb muscles during walking, which further contribute to the physiological congestion of the blood vessels in the nerve roots of the corresponding spinal ganglia in the spinal canal, followed by venous stasis and ischemic radiculitis due to the blockage of microcirculation in the corresponding area after the nerve roots are strained. When the patient squats, sits down, bends over or rests flat, the pressure load on the nerve root is reduced, the source of stimulation during muscle activity is eliminated, and the ischemic state of the spinal cord and nerve root is improved, so the symptoms are reduced and disappear. When walking again, the above-mentioned symptoms appear again, and then rest, the symptoms are relieved again, and so on, alternately, forming intermittent claudication. It is one of the main clinical features of lumbar spinal stenosis. How to identify intermittent claudication? Lumbar spinal stenosis manifests as intermittent claudication of neurogenic origin, which is sometimes confused with intermittent claudication of vascular origin (e.g. thrombo-occlusive vasculitis) and can be misdiagnosed. (2) Intermittent claudication of neurogenic origin may have segmental sensory deficits in the lower extremities, and intermittent claudication of vascular origin is a garter-type sensory deficit. (3) The walking distance of intermittent claudication of neurological origin is gradually shortened with the prolongation of the disease, while intermittent claudication of vascular origin is not obvious. (4) If necessary, arteriography is feasible, with good arteries in intermittent claudication of neurogenic origin, while intermittent claudication of vascular origin can show the arterial lumen narrowing area.