Epilepsy is indeed associated with genetics, including the main external causes of epileptogenesis. Epilepsy has a genetic predisposition, but this only means that people with genetic qualities have a low seizure threshold, increased susceptibility, and are prone to seizures when they encounter certain environmental factors, while the onset of seizures is determined by a combination of internal and external factors. In reality, only a small percentage of seizures are caused by genetic factors, so patients do not need to worry too much about the genetic aspects of epilepsy. Primary epilepsy (idiopathic epilepsy) is epilepsy for which no clinical cause can be found, and it is hereditary. Fifty percent of people with epilepsy start within 10 years of age, which means that children and adolescents are the most common. This is because children’s brains are in an immature stage of development and have their own unique physiological, anatomical, and biochemical characteristics. Because of the immaturity of the central nervous system, the nerve cells themselves are unstable, the release of neurotransmitters is unbalanced, and they are susceptible to stimulation and convulsions, so epilepsy (crohn’s disease) is mostly seen in children. Because of the age-dependent nature of its localization, degree of spread, and onset, there are many epilepsies and specific seizure types seen only in children. At the same time, because the pediatric brain is more plastic and the convulsions themselves are less damaging to the immature brain than to the mature brain, the vast majority of epilepsy can be treated as long as medication is adhered to and confidence is built to overcome the disease as the child grows and the brain matures.