Diabetic retinopathy (glucose retina) is a characteristic ocular complication of diabetes. All fundus changes are the result of diabetic retinal microangiopathy, and it is one of the four major blinding eye diseases, the incidence and blindness of which are increasing year by year. Currently, there is no effective treatment for diabetic retinopathy, and early diagnosis and aggressive treatment to slow its progression is essential to preserve some vision in the vast majority of patients. Most diabetic patients are not aware of the glycoplegia, and because the disease develops slowly in the early stages, the symptoms are insidious, and they feel good about themselves, it is difficult to see the doctor consciously. The best time to stabilize the condition is when interventions are made. Therefore, diabetic patients should be followed up regularly by an eye care professional from the time they are diagnosed. Once the early lesions are detected, early treatment is necessary to prevent the lesions from developing into advanced blindness and causing irreparable damage. It is now recognized that the disorder of glucose metabolism mechanism is an important reason for the occurrence of glucose reticulum, and the occurrence of glucose reticulum is greatly related to the duration of diabetes and the level of fasting glucose, i.e. the longer the duration of the disease and the poor control of fasting glucose, the greater the possibility of glucose reticulum. It is worth noting that blood glucose level is only a transient indicator reflecting the quality of blood glucose control, while glycosylated hemoglobin level is an average indicator reflecting the quality of long-term blood glucose control, which can reflect the blood glucose situation of the patient for 2 to 3 months before the test, and is not affected by short-term blood glucose fluctuations. A chronically high glycosylated hemoglobin indicates a greater probability of developing diabetic retinopathy in the future. Blood lipid levels are associated with the glucose network. Recent studies have found that total blood cholesterol and LDL are associated with the severity of retinopathy in patients with glucose retina, and that glucose retinal progression is associated with blood triglycerides and LDL. Glucose reticulum is associated with poor blood pressure control. Poor long-term blood pressure control is a contributing factor to glucose reticulum. Compared with non-diabetic patients, the rate of hypertension is 0.5 to 2 times higher in diabetic patients, and the degree of damage to the heart, brain, and kidneys from diabetes and hypertension is much greater than in patients with primary hypertension alone or diabetes alone. Patients with diabetes combined with hypertension not only have small vascular lesions, but also large vascular lesions, which are highly susceptible to cardiovascular and cerebrovascular diseases, and can accelerate the occurrence and development of renal lesions and retinopathy. The glucose network is closely related to smoking. Cigarettes, when burned, can release more than 4,000 ingredients, the main components are nicotine, tar, polycyclic aromatic hydrocarbons and carbon monoxide. Smoke also contains a variety of heavy metals and harmful minerals, such as aluminum, lead and mercury. Nicotine and some vasoactive substances can cause vasoconstriction, leading to ischemia and hypoxia in eye tissues; nicotine and carbon monoxide can make platelet aggregation increase, easy to thrombosis, which can cause eye vascular lesions. Reasonable and effective control of blood sugar can delay the occurrence and progress of glucose network, nephropathy and neuropathy. However, in the early stage of insulin application, it can cause pericyte contraction and blood flow reduction, resulting in retinal ischemia and hypoxia, and enhance capillary permeability, which promotes the progression of glucose reticulum, so the use of clinical insulin needs to be cautious. For glycoplasmic reticulum, drug treatment is generally only for early stage patients. Once exudation, hemorrhage, microaneurysm and neovascularization occur, laser photocoagulation treatment should be considered, and its effect is still better. However, laser treatment is not effective for all patients with glycogenic reticulum, and if used improperly, it may bring about serious adverse effects and accelerate the progression of the disease; in addition, even with proper laser treatment, it may cause some damage to vision, visual field and dark adaptation. Therefore, it is important to weigh the pros and cons of laser photocoagulation treatment for patients with glycogenic reticulum.