As the name implies, epilepsy caused after cranial trauma is known as traumatic epilepsy. Most craniocerebral trauma does not cause or leave behind seizures. The formation of epilepsy is related to the extent of the trauma, the location of the injury, and the duration of the injury, in addition to the patient’s own qualities. Generally the more severe the injury, the higher the incidence of epilepsy. Injuries to the motor areas of the cerebral cortex, the temporal lobe, and especially the medial aspect of the temporal lobe have a relatively high rate of epilepsy. Among patients who develop epilepsy after trauma, the highest rate of epilepsy occurs between 1 month and several years after injury, and less frequently in the immediate post-injury period. It is worth mentioning that craniosynostosis caused by childbirth is one of the leading causes of pediatric epilepsy, accounting for 8 to 11% of pediatric epilepsy. This is one of the more common types of epilepsy, and craniocerebral trauma often leaves behind adhesions and softening of the brain tissue. The epileptogenic focus is located at the periphery of the adhesions or softened brain tissue. The surgical approach is to remove the epileptogenic focus under EEG monitoring, and most patients have complete cessation of seizures.