Treatment methods and indications include antithyroid drug therapy, adjuvant therapy and life therapy to enhance nutrition. Anti-thyroid medication is based on thiourea compounds, and this method is the main method in internal treatment. Adjuvant treatment is mainly symptomatic treatment with insulin and reserpine. Life treatment is to take proper rest, give adequate nutrition and calories in diet, including sugar, protein, fat and B vitamins, and pay attention to avoid mental stimulation and excessive fatigue. Drug therapy uses thiourea drugs to inhibit the organic iodine in the thyroid gland and reduce the synthesis of thyroid hormones, but this type of drug does not inhibit the iodine uptake by the thyroid gland and the release of already synthesized hormones, then beta-blockers should be added at the early stage of treatment, such as take-home pay and betalactam. However, they must be taken for a long period of time, and the dosage can be gradually reduced over a period of about one and a half to two years until the drug is discontinued. However, about one-third to half of the patients will have recurrence, especially those with large necks or a diet with high iodine intake (such as frequent consumption of seaweed, seaweed, iodized salt). In addition, a small number of patients may experience drug allergies such as itchy skin, rash or reduced white blood cells (prone to fever and sore throat) and abnormal liver function within the first two or three months of taking the drug. If these phenomena occur, it is advisable to consult a doctor for further diagnosis and treatment. Indications for drug treatment: 1. mild and small Graves’ hyperthyroidism; 2. young (under 20 years old), pregnant women, old and weak or combined with serious liver, kidney or heart disease and not suitable for surgery; 3. preparation for surgery; 4. relapse after surgical treatment and not suitable for isotope treatment; 5. as adjuvant therapy to radioisotope treatment. The side effects of anti-thyroid drugs for hyperthyroidism treatment: propylthiouracil and tabazol can cause leukopenia, which usually occurs in the first few months after the drug is used. The most serious side effects of western antithyroid drug therapy for hyperthyroidism are leukopenia and granulocyte deficiency, which cause a significant decrease in systemic resistance due to low granulocyte count and subsequently lead to serious systemic infections, which are extremely life-threatening. Therefore, attention should be paid to the occurrence of granulocyte deficiency during the use of the drug, and if detected in time, the chances of cure are still relatively high. Granulocyte deficiency occurs mostly during the first 3 months of drug use, but can also be seen at any time after drug use. Therefore, special vigilance should be exercised during the first to third months of drug use. Once granulocyte deficiency occurs, antithyroid drugs should be stopped immediately and the patient should be sent to hospital for resuscitation. Because the patient’s resistance is too weak, he should be resuscitated in a sterile and isolated ward and treated with large amounts of glucocorticoids and antibiotics. After cure, the patient should not be treated with antithyroid drugs for hyperthyroidism.