Fungal keratoconus is a stubborn and highly blinding septic keratitis caused by fungal infection. In the past 20 years, due to the widespread use of antibiotics and corticosteroids, the incidence of fungal corneal ulcers has increased relatively and is no longer an uncommon disease. At present, the most reported domestic data for Aspergillus, followed by Fusarium, Candida, yeast, cephalosporium, etc. The vast majority of patients are farmers, although it can occur throughout the year, but mainly concentrated in the agricultural summer harvest and autumn harvest season. The most common corneal injury is crop injury, but also nail gouge, can also be seen in other nature of keratitis secondary to fungal infection. It is believed that long-term use of corticosteroids or antibiotic eye drops can easily lead to fungal infections, and in recent years there have been more and more cases of contamination from wearing corneal contact lenses, while low systemic or local immunity is also a factor in the development of the disease. Clinically, different pathogenic species can have different clinical manifestations, but the common symptoms are slow onset, mild irritation symptoms, early ulcers are shallow in nature, the surface is covered by gray or creamy white “mossy” material, dry and less glossy appearance, slightly elevated, “mossy The “mossy” material is easy to pick out, the stroma has mycelial multiplication, the infiltrate is more dense, the border is pseudopod because the mycelium extends around, forming a pseudopod, and the distribution is somewhat cloudy at its periphery, forming the so-called satellite lesions. Sometimes shallow grooves appear at the edges of the infiltrate due to collagen lysis. As the infiltrate progresses deeper, the tissue is necrotic and falls off, forming an obvious ulcer. As fungal toxins invade the anterior chamber, they cause iritis and an accumulation of pus in the anterior chamber, which in advanced stages is viscous and often contains fungus. The ulcer can eventually penetrate and cause endophthalmitis. The entire course of the disease is slow and can take up to 2 to 3 months. The fungal hyphae have the ability to grow deeper, so it is easy to recur, and sometimes the ulcer heals initially, but then recurs. The diagnosis is usually confirmed by smear examination of the fungus or repeated culture of the fungus. Because of the low detection rate of smear and the long culture time, the possibility of fungal ulcers should be considered first in the case of ulcers that occur after crop trauma, especially in cases with less acute onset, and active treatment measures should be taken if multiple drug treatments are ineffective. Fungal corneal ulcer treatment is based on antifungal drugs, commonly used 0.25% dicloxacillin B (the drug subconjunctival injection is prone to conjunctival necrosis, clinical attention should be paid to), chrysomycin ophthalmic ointment, mycobacterial ophthalmic ointment and 10% Dafukang, 1% miconazole, 1% clotrimazole, etc., 3 to 4 times a day, and systemic application of antifungal drugs. Corticosteroids are contraindicated whether used systemically or topically alone. As the disease causes iritis, frequent attention should be paid to dilating the pupil.