The normal value of intraocular pressure is between 10 and 21 mmHg. When the IOP measurement is higher than normal, the most common cause is glaucoma. Other causes are: hypertension; and errors in IOP measurement due to thick corneas and poor patient cooperation during the examination are also possible causes. Glaucoma, an eye disease that eventually leads to optic nerve damage due to pathologically high intraocular pressure, includes both primary and secondary. A single measurement of IOP higher than normal does not easily lead to the conclusion that the patient must have glaucoma. The diagnosis of glaucoma needs to be made by a comprehensive analysis combining multiple IOP measurements, visual field examination and fundus examination. Some chronic glaucoma requires dynamic observation for one or two years to confirm progressive damage to the visual field and progressive enlargement of the fundus C/D before the diagnosis can be confirmed. Glaucoma is a disease that can lead to blindness, and once diagnosed, it requires medication or surgery to control the IOP and slow down the progression of the disease. In hypertelorism, the patient’s IOP is higher than normal but never shows glaucoma-like damage to the optic nerve. It is mostly seen in developing adolescents and presents with only high IOP, which is dynamically observed for several years without glaucoma manifestations such as visual field defects and fundus C/D enlargement, and does not require treatment and does not cause damage to the eye. However, to avoid misdiagnosis, it must be differentiated from glaucoma, so dynamic observation for several years is also required. Measurement errors, too, may be the cause of high IOP measurements. Some people have thicker corneas, even up to 600 microns or more, and IOP values measured by a non-contact tonometer can be high. In this case, a corneal thickness measurement should be performed at the same time as the IOP measurement, and the measured IOP value should be corrected according to the thickness of the cornea. Sometimes, the patient’s IOP measurement may be high due to tension, squeezing of the eyes, pressure on the eyeball when the examiner helps to pull the eyelids apart, and instrument measurement errors, which should be standardized during the measurement, allowing the patient to rest sufficiently and measure in a relaxed state, and the goods should really be replaced with an IOP meter of a different principle, such as a flattening IOP meter under surface anesthesia. If the IOP is still higher than normal after the above adjustments, glaucoma should be investigated. In summary, there are many causes of high IOP, both real and pseudo, as well as disease and non-disease. Patients should go to the ophthalmology department for detailed examination and clear diagnosis, and then for targeted treatment. With advances in medical technology, the vast majority of retinal detachments are treatable. Depending on the type of retinal detachment, the extent of the detachment, and the length of the detachment, the treatment varies. And the final vision of the patient varies. The most common type of retinal detachment is a foraminal retinal detachment due to a fissure in the retina, which must be surgically closed. In the case of traumatic retinal detachment, spontaneous retinal detachment due to high myopia, retractive retinal detachment due to fundus hemorrhage and vitreous hemorrhage, the retina must be surgically repositioned by intraocular pneumatization, vitrectomy, and injection of silicone oil or heavy water. In exudative retinal detachment, no fissure appears, but the retina is detached due to other ocular diseases, so the main focus is on treating the primary disease and surgery is usually not required. When a retinal detachment is treated and reset, it is a return of the anatomical position, which does not mean that the visual function will be similarly restored to the pre-detachment level. This is because the photoreceptor cells of the retina, during the pathological process of detachment, undergo irreversible death. If the detachment is small and does not involve the macula, and if the detachment is short, better vision can be restored after surgery. Therefore, the only way to expect to be able to restore the best possible vision is to make a timely diagnosis, cooperate with treatment, and allow the retina to be reset in the shortest possible time.