The first one is the erectile dysfunction of the male penis, which is abbreviated as ED. It is manifested as the difficulty of sexual intercourse when the penis cannot be erected or can be erected but is not hard. The first is divided into ED caused by diseases of the reproductive system itself and diseases of the fee reproductive system; the second is divided into ED caused by non-organic lesions and organic lesions. the following is the second classification method. 1, non-organic lesions that the cerebral cortex on the inhibition of sexual excitement and the spinal cord erectile center hypoexcitability. Also known as functional reasons, no organic lesions exist, can be over-indulgence, so that the nervous system is often in a state of hyperexcitation, and eventually exhausted by overexcitation. It can also be due to chronic illness, physical weakness or overexertion, physical or mental labor caused by physical weakness or neurasthenia. Another reason is from the mental factors, such as fear of pregnancy, sexual intercourse environment, couples with cold feelings, or thought that the former seminal emission, masturbation and worry about sexual function problems, or even out of the lack of knowledge of sex and fear or fear of female blame caused by the psychology. 2, organic causes impotence caused by old age, organ system lesions, drug effects or surgery, such patients can not get an erection even in the case of strong sexual stimulation. The causes of organic impotence are: (1) Inadequate blood flow: Atherosclerosis or other vascular lesions can lead to inadequate blood flow. Atherosclerosis, if it occurs in the arteries supplying the penis or in the vessels governing nutrition, can also cause impotence in patients. (2) Nerve disorders: such as spinal cord injury, spinal cord transection, spinal cord tumor, temporal lobe lesions, can cause impotence due to nerve disorders that conduct sexual excitement. (3) Endocrine disorders: mainly diabetes mellitus, hypothalamic-pituitary abnormalities and primary gonadal insufficiency: ① Hypothalamic pituitary abnormalities, which account for about of impotent patients, are mainly pituitary tumors, others have peripheral focal infiltrations or pituitary blood flow disorders, etc. Factors contributing to impotence are decreased luteinizing hormone or follicle-sparing hormone (LH and FSH) due to decreased crude gonadotropin-releasing hormone (GnRH) and increased prolactin; ② diabetes mellitus accounts for approximately. Impotence occurs in patients, 2 to 5 times more than the normal population of the same age. If there are peripheral and vegetative neuropathy and vascular abnormalities can aggravate impotence, the degree of impotence is generally not consistent with the degree of diabetic vascular retinopathy. Impotence is mainly caused by parasympathetic neuropathy in the perineum caused by diabetes mellitus; reduced blood flow to the penis due to arterial stenosis caused by diabetes mellitus, and mental factors caused by metabolic disorders caused by diabetes mellitus; hyperthyroidism, impotence can occur in about half of hyperthyroidism, but the mechanism is not clear; hypothyroidism, this disease causes a decrease in the secretion of thyroid hormones. Testosterone and testosterone-binding globulin are reduced and prolactin is increased. The cause of impotence is related to degenerative lesions of the testicular varicoceles due to systemic protein synthesis disorders and a decrease in interstitial cells. (6) Cortisolism, bilateral adrenal cortical hyperplasia, adenoma, adenocarcinoma or medically induced cortisolism can inhibit gonadotropin and testosterone secretion by testicular interstitial cells and cause impotence; (7) Adrenal insufficiency is related to wasting and malnutrition, resulting in a decrease in LH secretion by the pituitary gland and testosterone secretion by testicular interstitial cells; (8) Feminizing tumors, such tumors can cause impotence. Feminizing tumors, such tumors can occur in the adrenal glands or testicular mesenchymal cells, and laboratory tests have increased estrogen changes, leading to gynecomastia and impotence caused by testicular atrophy. 3. Other organic diseases, including: (1) surgical complications, such as transcervical prostatectomy, sympathectomy, radical rectal cancer, which can affect the anatomy and physiology of erection and cause impotence; (2) drug effects, such as guanethidine, reserpine, methyldopa, progesterone, anticholinergic drugs, estrogens, etc.; (3) inflammatory lesions; (4) toxic substances, such as lead and (5) trauma, such as testicular trauma castration, pelvic fracture, penile trauma, etc.; (6) urological diseases, penile sclerosis, scrotal effusion, varicocele, prepuce, scrotal erosion, etc. may cause impotence.