What causes bladder spasms?

  Cystospasm is a spastic contraction of the bladder smooth muscle or bladder sphincter, without inflammatory changes. The clinical presentation is characterized by dribbling urine, temporary shutdown and urinary abdominal pain.  Etiology: 1. Anatomical structure.  The female urethra is short and straight, and the external urethral opening is commonly deformed, such as the hymenal umbrella and the fusion of the urethral hymen; and there is a large number of bacteria in the perineum, and as long as there are causative factors of infection present, such as sexual intercourse, catheterization, unclean personal hygiene, and individual resistance to bacteria is reduced, the infection can be roughly upstream. Rarely, it is caused by blood-borne and lymphatic infections.  The male urethra is a muscular mucosal tube, about 20 cm long, which can be divided into two sections, front and back, bounded by the urogenital diaphragm, with two bends, subpubic and prepubic, so it is generally not easy to cause an upstream infection, often secondary to other lesions, such as acute prostatitis, benign prostatic hyperplasia, prepuce, urethral stricture, urinary calculi, kidney infection, etc., but also secondary to infection of adjacent organs such as peri-appendiceal abscess.  2, estrogen deficiency.  The frequent occurrence of urinary tract infections in menopausal women is due to the decrease in lactobacilli in the vagina caused by the lack of estrogen and the increase in the reproduction of pathogenic bacteria is an important factor in the infection.  3, the causative bacteria most of the pathogenic bacteria is E. coli.  Other rare ones are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Streptococcus faecalis and Staphylococcus aureus. In children, adenovirus infection can lead to hemorrhagic cystitis, but viral cystitis is rare in adults.  Pathologic changes are more common in superficial cystitis, most evident in the internal urethral opening and bladder triangle. In the early stages of acute cystitis, the bladder mucosa is congested, edematous, dark red, and infiltrated with leukocytes; in the later stages, the mucosa becomes more brittle and prone to bleeding, with a granular surface and a localized superficial ulcer or pus moss covering it with exudate, usually without involvement of the muscularis. Inflammation has a tendency to heal spontaneously and leaves no traces after healing. If treatment is incomplete or if there is a foreign body, residual urine, or upper urinary tract infection, it tends to become chronic. No traces are left behind. If the treatment is incomplete or there is foreign body, residual urine, upper urinary tract infection, etc., it is easy to turn chronic.