An important factor in the pathogenesis of cervical hypertension is the chronic injury to the cervical spine and surrounding soft tissues secondary to cervical spondylosis and cervical spine syndrome, the extrusion of nerves and blood vessels, the pulling stimulation of cervical sympathetic nerves and cervical arteries, increased excitability of cervical sympathetic nerves, vasospasm cerebral ischemia, reflexive secondary thalamic constrictive vascular center with enhanced lateral reticular pressor, making the caliber of blood vessels smaller, increased peripheral resistance, and increased blood pressure. The author treated eight patients with cervical hypertension without a history of primary hypertension and without other secondary hypertensive disorders with small needle closed surgery, manual repositioning, and periarticular drug injection with satisfactory results. The lateral medullary reticular formation, the thalamus, and the cortical centers are the motor centers that regulate vasodilation and contraction. The pathogenesis of cervical hypertension comes from cervical spondylosis or cervical spine syndrome, and the combination of these two diseases is an important factor in the occurrence of cervical hypertension. However, not all patients with damage to the cervical spine have hypertension. Only the affected lesion site, squeezing nerves and blood vessels, enhanced intravascular pressure stress and increased peripheral resistance of blood vessels can lead to increased blood pressure. The main symptoms of the eight patients in this treatment group were upper cervical manifestations: vertigo and stiff neck pain often accompanied by headache, tinnitus, hearing impairment, and difficulty seeing objects. The pathological changes included sclerotic disc protrusion, bony bulge, joint misalignment and ligamentous loosening and distortion, ligamentous hypertrophy, calcification and ossification, adhesions, muscle spasm, and sensory pain. These symptoms stimulate the supracarotid sympathetic ganglion, resulting in increased excitability of the internal carotid nerve and vertebral artery nerve, leading to an increase in impulses from the thalamic constrictor center and the lateral pressor area of the reticular formation, decreased secretion of vasopressin, sympathetic excitation, vasospasm, smaller caliber, and increased blood flow resistance to hypertension. Sympathetic ganglion fibers form the heart’s plexus and are distributed to the sinus node and coronary arteries, so when sympathetic excitability is increased, the heartbeat is accelerated and the coronary arteries are diastolic, resulting in increased blood pressure. Key issues of treatment (treatment of 8 patients). We feel that the effect is not in the advanced treatment means, but in the determination of the main factors leading to cervical hypertension; and then according to the form and degree of pathological changes in the main factors to choose the appropriate acupuncture therapy, manipulation and drug therapy, the only way to change the symptoms is the main factors. Needle knife, stripping adhesions, sclerosis, contractures; manipulation, pushing the oblique spinous process, changing the relationship of the posterior joint while changing the relationship of the vertebral artery of the hook. The posterior joint was restored to normal, and the posterior joint capsule was also restored to normal, relieving the compression and stimulation of the posterior branch of the spinal nerve, and also relieving or alleviating the compression and stimulation of the sympathetic nerve fibers or sympathetic nerve trunks by the joints and soft tissues, restoring the balance of both cervical segment stress and vascular nerve stress, and lowering the blood pressure to normal. Drugs: saffron, compound salvia, adenosine triphosphate, tretinoin, lidocaine mixture can eliminate and relieve the inflammatory tissues on the sympathetic nerve and vertebral artery stimulation, invigorate blood circulation, relieve blood stasis, promote cell metabolism, increase the effective blood volume of vertebral artery and basilar artery to eliminate carbon dioxide.