Gluteal muscle contracture (GMC) is a clinical syndrome caused by degeneration and contracture of the gluteal muscle and its fascial fibers from various causes, resulting in a characteristic gait and signs of limited hip function, but the etiology and classification are not well understood. Etiology 1, injection factors hip contracture is closely related to the hip receiving repeated intramuscular injections. The injury factors include mechanical injury and chemical injury, etc. Most scholars believe that chemical injury from injectable drugs is the main etiology. 2, susceptibility factors immune factors; scar body. 3, trauma, infection and other factors congenital hip dislocation postoperative complications; gluteal myofascial interval syndrome sequelae; hip infection. 4, genetic idiopathic factors. Clinical manifestations of the disease are more common in children, more males than females, and mostly bilateral. 1, hip dysfunction patients with limited internal rotation and internal retraction of the hip joint activities. When standing, the lower limbs are externally rotated and cannot be fully brought together. Walking often have external eight, swaying gait, quick step is jumping state. When sitting, the legs cannot come together, the hips are separated in frog position, and it is difficult to rest one thigh on the other thigh (cross-leg test). When squatting, the lighter person squats with both knees apart, then squats and then comes together (circle test). In heavy cases, the hip joints can only squat in the abducted and externally rotated position, and the knees cannot come together when squatting, and the heels do not touch the ground, showing a frog-like pattern. On physical examination, it can be found that there is skin depression in the upper part of the outer hip, and the depression is more obvious when the hip is inwardly retracted, and there is a sense of tightness in the hip, and the lower limb is in the abducted and externally rotated position, and the hip is restricted in internal retraction and internal rotation, and the hip flexion is restricted in the neutral position of the lower limb, and the affected hip must be abducted and externally rotated, so that the affected side of the hip can draw a semicircle outward before it can be fully flexed back into the original sagittal plane. Ober’s sign is positive. 2.Pelvic variant with long duration and severe disease may have the acetabular floor convex to the pelvis, forming Otto’s pelvis. In children with gluteus medius contracture, there is hypertrophy of the greater trochanteric epiphysis. Children with bilateral asymmetric gluteal contracture may have a tilted pelvis and secondary scoliosis of the lumbar crest. The anterior superior iliac spine is lower on the severe side than on the light side, and the umbilical-ankle distance is longer on the heavy side than on the light side, while the distance from the greater trochanter to the ankle is equal on both sides. Examination 1.X-ray few pelvic and hip joints have secondary changes, such as, hip subluxation, etc. 2.Laboratory tests are mostly abnormal. Treatment of this disease mostly requires surgical treatment of hip contracture band severance plus gluteus maximus stop release, which uses a curved incision above the posterior aspect of the greater trochanter to expose the posterior edge of the broad fascia, the lower edge of the hip contracture band and the lower part of the gluteus maximus tendon plate, with small surgical incisions and trauma, and can fully address the causative factors in the operative field, with satisfactory results.