There are more treatments for warts, but the recurrence rate is as high as 60% to 70% or so, for the following reasons.
1, after infection with human papillomavirus, latent in the squamous epithelium, migratory epithelial cells to reproduce, replication, warts in batches, early warts treatment to clear, and then the emergence of new warts.
2, atypical warts or subclinical infection is not dealt with.
3, the occurrence of warts after their own treatment or because of contact with the occurrence of self-inoculation.
4, infected with a variety of types of human papillomavirus, some types of virus is easy to recurrence of the type.
5, infected with human papillomavirus to a spouse or sexual partner, and then infected after the cure.
6. Re-infection with HPV from other sexual partners.
7.Patients combined with other STDs, local inflammatory stimulation prompted the spread of HPV.
8, their own resistance to reduce.
What should I pay attention to after treatment of warts?
The pathogen (human papillomavirus) of condyloma acuminata is more active, so the following aspects should be noted after the disease.
1, check if your sexual partner has warts.
2, check for other STDs, such as gonorrhea, genital herpes and mersin.
3, emphasizing early diagnosis and early treatment.
4, to avoid reducing their resistance to various unfavorable factors.
5.Prohibit sexual intercourse during the period of illness.
6, pay attention to rest, strengthen nutrition, prohibit smoking, alcohol and eat less spicy and sour food, less strong tea and coffee.
7.Protect local cleanliness, clean and dry.
8, household items should be used and stored separately to avoid misuse by others.
The causes of the onset and recurrence of genital herpes
Genital herpes is mainly caused by herpes simplex virus type II. During sexual contact, virus particles invade the keratinocytes of the skin and mucous membranes through tiny fissures, where they multiply, replicate, and spread to surrounding cells. At the time of initial infection, the body does not yet have antibody formation and is susceptible to viraemia and infection of other internal organs. When neutralizing and complement-binding antibodies are produced in the body, viremia and disseminated spread of the virus are suppressed, but the virus in local cells is difficult to eliminate and virus multiplication cannot be stopped. These viruses can be transferred via peripheral nerves along the nerve axis into the sacral ganglion where they remain latent for a long time, which is the underlying cause of recurrent genital herpes after treatment.
What are the factors that affect the recurrence of genital herpes?
1, is not to get scientific diagnosis and treatment, some patients in the treatment process there is contempt, impatient psychology, not according to the course of medication or their own indiscriminate use of drugs buy drugs, resulting in the occurrence of drug resistance and physical toxic side effects.
2, is not thoroughly treated. The current genital herpes routinely used anti-viral drugs and chemotherapy only treats the symptoms but not the root cause, and so far there is no effective drug for complete eradication. The use of interferon, thymidine, interleukin-2 and other treatments can temporarily inhibit viral replication, reduce or prolong the time of recurrence, can not completely remove the presence of blood tissue cell nuclei in the virus particles, after stopping the drug is very easy to relapse.
3, is the existence of their own inoculation infection or re-infection, due to poor personal hygiene, sexual partners did not timely examination and treatment and re-infection resulting in recurrent attacks.
4. It is the presence of low immunity of the organism, individual differences, and recurrence at the damage after the herpes has subsided by treatment.
The solution to the recurrence of the above two diseases
It is necessary to visit the hospital regularly for follow-up observation for 3-6 months, to overcome impatience, to treat scientifically as required and to eliminate unfavorable factors in order to achieve clinical cure.