First, self-limiting diseases and TMD clinical self-limiting phenomenon self-limiting disease, that is, the disease in the development of a certain degree can automatically stop, only symptomatic treatment or no treatment, rely on their own immune or repair ability can gradually recover from the disease. The prevalence of TMD is very high, Agerberg (1975) conducted a survey on 1106 people, the prevalence of TMD is 40%, Solberg (1979) conducted a survey on 739 people, the prevalence is 76%. In a survey of 1,321 people by Xu Cherry (1985), 13.1% were positive for subjective symptoms and 75.78% were positive for objective signs. Shi Zongdao (2008) reported that 30% of the population had some symptoms of TMD and 65% of the surveyed population had symptoms or signs of TMD. Although the prevalence is high, not many patients actually develop severe symptoms. Furthermore, disc displacement is not always clinically symptomatic.Katzberg (1996) found 33% (25/76) of those without symptoms of TMD to have disc displacement by MRI.Kuita (1998) found that after 2.5 years of untreated follow-up of 40 patients with MRI-confirmed irreversible anterior disc displacement, 43% of these patients were asymptomatic, 33% had symptomatic relief, and only 25% had no improvement in symptoms or needed to be followed for 2.5 years. percent of patients had no improvement in symptoms or required treatment. Sato (1997) in 44 cases of irreducible anterior disk displacement without treatment, the follow-up results were significant pain reduction after 6 months, significant improvement in tensor digitorum at 12 months, and joint compression pain after 18 months in only 2 cases i.e. 9.1%. These phenomena indicate that TMD is a self-limiting disease. According to Shi Zongdao, the annual self-resolution rate of TMD in the natural population is 42.9% for symptoms and 37.6% for signs. Histologic basis for the self-limiting nature of TMD The cartilage of the mandibular condyle is secondary cartilage, which after development is complete still has the function of multidirectional differentiation to adapt to the needs of development and tissue remodeling, so the temporomandibular joint (TMJ) is a synovial joint with the ability of adaptive remodeling. Adaptive remodeling of the articular region can also occur after anterior displacement of the TMJ disc, which is manifested by morphological and structural changes affecting the condylar cartilage, and the appearance of structures such as chondrocytes and dense connective tissue in the biplatonic region of the articular disc. The biplatonic zone is a loose connective tissue located behind the articular disc and is not loaded under normal conditions. After displacement of the articular disc, the bilaminar zone is pulled forward over the condylar process and is subjected to abnormal loading, which firstly results in destructive changes, manifested by synovial membrane destruction and collagen fiber breakage. Then the repair phenomenon occurs: chondrocytes appear in the biplatysmal area and express type II collagen and proteoglycan polymer mRNA, which are unique to chondrocytes, and synthesize cartilaginous ECM, which becomes similar to fibrocartilaginous tissue. This structure is biomechanically resistant to compression, friction, and shear forces, and helps the biplate region to withstand joint loads. Whether the biplate area can undergo effective adaptive remodeling to form fibrocartilage and exercise the role of the articular disc after TMJ disc displacement may be the key to whether TMD can be self-healed. Third, the treatment strategy of TMD This self-healing or self-limiting characteristic of TMD requires us to be careful when choosing treatment options. Studies have shown that a significant proportion of patients, through therapeutic education and simple symptomatic management can eliminate the symptoms; some patients just have certain concerns about their disease; some signs and symptoms also do not cause progressive lesions and affect the patient’s quality of life, such as popping and ringing. How to determine if treatment is needed? The principle should be that the pain does not resolve, affects the quality of life, or has progressive pathologic changes already present. The AADR strongly recommends that unless there is a clear and justifiable indication, treatments for TMD should first and foremost be those that are conservative, reversible, and based on evidence-based medicine. Although no treatment has been proven to be consistently effective, many conservative treatments are at least as effective as those that are invasive in relieving symptoms, and these conservative treatments do not lead to irreversible changes, greatly reducing the chances of leading to new injuries.