Post-stroke patients often have dysphagia, a swallowing disorder that can lead to higher healthcare costs as well as higher rates of complications such as dehydration, malnutrition, and pneumonia. Researchers have found that transcranial direct electrical stimulation improves the treatment of dysphagia after stroke. tDCS is a treatment that stimulates affected brain regions with weak electrical currents. This study included 16 patients with dysphagia after acute stroke. Their dysphagia manifested itself in the form of reduced tongue movement, coughing and choking during eating, and vocal cord paralysis. The patients underwent ten 30-minute swallowing sessions and were randomized into treatment and control groups. In each group, electrodes were placed on the scalp surface in the area of the stroke-damaged hemisphere associated with swallowing. During the first 20 minutes of treatment, the treatment group received tDCS followed by 10 minutes of swallowing training. In the control group, the DC current became smaller and was cut off after 30 s. The control group received tDCS for the first 20 minutes of treatment. Patients were assessed before the trial, at the end of the trial, and three months after the trial. Patients in both groups underwent PET scans before and after treatment to show the effects of treatment on brain metabolism. None of the patients showed signs of discomfort or fatigue during the treatment. There were no significant differences between the two groups in terms of age, gender, stroke site, or degree of brain damage. Post-treatment assessment revealed that dysphagia improved in all patients and there was no significant difference in the degree of improvement between the two groups. However, at the 3-month follow-up period, the treatment group showed significant improvement over the control group. In the PET study, PET scans of patients who underwent tDCS showed a significant difference in brain metabolism before and after treatment. Although tDCS was performed only in damaged brain regions, an increase in glucose metabolism was observed in uninjured brain regions, suggesting that tDCS may activate a large cortical network associated with swallowing recovery rather than simply affecting only the stimulated region. tDCS enhances the therapeutic effect of dysphagia after stroke. As with all previous studies, we need to enroll more patients in further studies to confirm the results of our trial. To maximize long-term efficacy we must determine the appropriate intensity of treatment as well as the duration of treatment.