After the operation, we returned to the ICU, closely monitored vital signs, observed drainage flow, and actively replenished blood volume medication. Perform symptomatic treatment such as cardiac strengthening, coronary expansion, anticoagulation, anti-infection, and myocardial nutrition. Dynamic monitoring of chest X-ray, blood gas, liver and kidney function, etc. The condition gradually improved, and the tracheal intubation was removed on the 2nd postoperative day. The patient returned to the ward on the 3rd postoperative day with sudden drop in blood pressure and atrial fibrillation, and was actively treated with cardioplegia, diuretic, coronary expansion and IABP. The dressing on the surface of the incision was slightly exuding and was relatively intact and dry. The patient’s chest wound was healing well. The patient’s chest wound healed well. The repeat cardiac plain film, echocardiogram and electrocardiogram did not show any abnormality, and the blood routine and other laboratory tests were normal. The patient was discharged 11 days after surgery. Corrected transposition of the great arteries is a rare form of congenital heart disease. If the central canal bends to the left during development, the dissected right ventricle is located posteriorly to the left and becomes the ventricle of the arterial system, which is called the left loop, while the dissected left ventricle is located anteriorly to the right and becomes the ventricle of the venous system. The arterial trunk remains separated and rotated, with the ascending aorta located anteriorly to the left and the main pulmonary artery located posteriorly to the right, with blood flowing from the right atrium through the mitral valve to the pulmonary artery and from the left atrium through the tricuspid valve to the aorta. In this case, the patient with corrected aortic transposition had an orthogonal atrium, left ventricular loop, and the aorta was located anterior to the pulmonary artery, and the blood flow was physiologically corrected without congenital anomalies such as atrial septal defect, ventricular septal defect, or patent ductus arteriosus. It is a functional corrected transposition of the aorta. In this case, the patient with corrected transposition of the great arteries was in middle age, and because his anatomic right heart assumed the function of the anatomic left heart, the weak anatomic right ventricular wall could not fully compensate for the function of the left heart due to the long-term high pressure situation in the arterial system. The anatomic right ventricle can develop cardiac insufficiency over time with increased ventricular volume load, leading to valvular closure insufficiency. Also patients with anatomic left ventricular gyral branch occlusion result in ischemic necrosis of the myocardium at its blood supply and segmental abnormalities of the myocardial motor order, further reducing the level of cardiac function. Patients with functional corrected transposition of the great arteries have physiologically corrected blood flow and can survive without treatment. However, the anatomical right heart assumes the function of the anatomical left heart, and the anatomical characteristics of the anatomical right heart determine that it cannot fully perform the function of the left heart, and as the heart function decreases, the patient eventually dies of heart failure. In patients with corrected transposition of the great arteries, the left and right branches of the atrioventricular conduction bundle are still distributed to the corresponding anatomic ventricle due to the change in the position of the ventricle, so the conduction bundle is also reversed. The original conduction bundle from the sinus node through the atrioventricular node to the ventricle is twisted through the ventricle, and its length increases accordingly, making it prone to atrioventricular conduction block. This is illustrated by the presence of left bundle branch conduction block in this patient. The patient with corrected aortic transposition in this case had valvular regurgitation and needed mitral valve replacement. Due to its own special anatomy, it is easier to replace the biologic valve than the mechanical valve, and it is appropriate to use a porcine biologic valve with a shorter pedicle; a bovine biologic valve with a longer pedicle would touch the posterior wall of the left atrium and could lead to ventricular rupture. The patient with functional corrected transposition of the great arteries had a gyral branch occlusion combined with mitral valve closure insufficiency because of the special case of corrected transposition of the great arteries, in which the right ventricle was used as the left ventricle, unlike the simple infarction combined with mitral valve closure insufficiency, with cardiac insufficiency and more likely to have circulatory instability after surgery. In this case, cardiac insufficiency appeared on the third postoperative day, and after active resuscitation with a lot of positive inotropic drugs and IABP assistance, cardiac function improved, and the condition improved, and the patient was discharged from the hospital. It indicates that the use of bypass graft combined with mitral valve replacement is an effective treatment for patients with corrected transposition of the great arteries combined with heart attack.