Polycystic ovary syndrome is an endocrine syndrome with a multi-causal and polymorphic pathogenesis (characterized by persistent anovulation, hyperandrogenism, and insulin resistance), involving multi-systemic reproductive and metabolic disorders. Diagnostic criteria: 1. Clinical symptoms: scanty menstruation or amenorrhea, absence of ovulation in 2 menstrual cycles as determined by basal body temperature. 2, hyperandrogenic syndrome: hirsutism, seizures O, or biochemical indicators LH/FSH>=2, elevated testosterone T, and exclude diseases such as Cushing’s syndrome and abnormal thyroid kinetics. 3, Ultrasound shows unilateral and/or bilateral ovaries with >10 follicles in a single section, 2-8 mm in diameter, with cystic changes. 4, T exceeds the normal value. Polycystic ovary syndrome is mostly characterized by cystic enlargement of bilateral ovaries combined with immature follicles or follicular atresia. As multiple follicles develop simultaneously and do not reach maturity, the estrogen level increases and the endometrium continues to proliferate. If the follicles are not well developed, the luteal development after ovulation is poor and the progesterone level is low, which affects the endometrial tolerance and makes conception difficult.