Facial myoclonus is a relatively common cranial nerve disorder with a large population of sufferers nationwide. Facial myospasm is mainly manifested as paroxysmal involuntary twitching of one side of the face. In most cases, facial myospasm tends to start with eyelid twitching, and as the condition further develops, it gradually extends to twitching at the corners of the mouth and nose, and even twitching and jumping of the entire half of the face, and some more severe patients may have twitching of the neck muscles as well. This frequent onset of facial muscle twitching will make the patient very tortured, painful, and seriously affect the patient’s normal work life and study. Many people wonder what causes facial muscle spasms. Medical research shows that the most important cause of facial muscle spasm is the compression of the facial nerve root by the surrounding blood vessels, which leads to demyelination of the facial nerve, so that nerve dysfunction, the facial muscles it innervates will occur abnormal movements and symptoms of twitching and jumping of the face. The responsible vessels compressing the facial nerve root are mainly the anterior inferior cerebellar artery and posterior inferior cerebellar artery, while the superior cerebellar artery is less common; in addition, the superior vagus artery and other variant large arteries such as vertebral artery and basilar artery may also form compression on the facial nerve leading to area spasm. Therefore, in order to cure facial spasm, we should start from the root of the disease, which is the compression of facial nerve roots by blood vessels, and achieve the treatment effect by releasing the abnormal compression between blood vessels and nerves. The current standard treatment for facial spasm is microvascular decompression. This minimally invasive surgery starts from the cause of facial nerve root compression by carefully exploring the vascular-neural anatomical relationship around the facial nerve root through surgical procedures, finding the responsible vessels that are compressing the facial nerve and isolating them, so that the facial nerve will no longer be compressed, stimulated and disturbed, restoring the normal function of the nerve and thus curing the disease with excellent results.