When blood pressure is significantly elevated in the acute phase of cerebral hemorrhage, appropriate blood pressure lowering is an important measure to prevent further bleeding. Acute hypertension in cerebral hemorrhage reflects the degree of pre-existing hypertension on the one hand and the cerebrovascular autoregulation mechanism to maintain normal cerebral blood flow under cranial hypertension on the other. Therefore, the principles of management of hypertension in the acute phase of cerebral hemorrhage are: (1) Patients with chronic hypertension have adapted to high blood pressure levels, and excessive hypotension disrupts cerebrovascular autoregulatory mechanisms, which can lead to hypoperfusion or cerebral infarction. (2) Patients with atherosclerosis may have local stenosis of cerebral blood vessels, and excessive blood pressure lowering may significantly reduce blood flow. (1) In acute cerebral hemorrhage, increased intracranial pressure and cerebral edema can aggravate hypertension, and dehydrating agents should be used first. If blood pressure still does not fall, it means that the increase in blood pressure may not be related to high cranial pressure, and then antihypertensive drugs should be selected. 2, antihypertensive drugs should be slow to prevent individuals from being abnormally sensitive to antihypertensive drugs. The seventh report of the Joint National Committee on Prevention, Detection, Evaluation and Treatment of Hypertension (JNC-7) states that blood pressure should be controlled at 160/100 mmHg after stroke.