Pulmonary valve stenosis is both the stenosis of the pulmonary artery orifice, accounting for 5%-8% of congenital heart disease, 90% of which is the stenosis of the valve, that is, the thickening of the three pulmonary valves, the junction fused heart contraction, due to the valve can not open, and the garden top sign. Heart murmur is the main sign, electrocardiogram shows right ventricular hypertrophy, cardiac X-ray shows little pulmonary blood, right heart enlargement, vertical-like dilatation of the pulmonary artery segment, echocardiography can be clearly diagnosed. Pulmonary stenosis can be classified as mild, moderate or severe according to the pressure difference between the right ventricle and the pulmonary artery. A pressure difference of 0.75 kPa (more than 10 mm Hg) can be diagnosed as pulmonary stenosis; a pressure difference of 10-50 mm Hg is considered mild stenosis; a left ventricular systolic pressure level of 50 is considered moderate; and a pressure level greater than the left ventricular systolic pressure level is considered severe. Except for severe pulmonary stenosis, the clinical symptoms in children are mild, and sometimes there may be shortness of breath after exercise. In severe cases, the right ventricular and right atrial pressures are elevated, causing the foramen ovale in the interatrial septum to open and blood from the right atrium to flow into the left atrium, resulting in bruising of the lips and fingernails. Patients with severe stenosis may also have heart failure, such as swelling, ascites, and hepatomegaly. Treatment of pulmonary stenosis currently includes surgery and balloon catheterization. Dilatation tears the adherent pulmonary valve and solves the stenosis problem, which is basically successful except for a few cases of failure, and the restenosis rate is extremely low, so it basically replaces surgery at present. Surgery may be required only in cases of valve dysplasia, small annulus, or in combination with other cardiac malformations.