Femoral head necrosis is one of the more common diseases in orthopedic clinics, and the first cause of femoral head necrosis is the use of hormones. The occurrence of femoral head necrosis is related to the dose and route of hormone intake, and the incidence of femoral head necrosis is higher for those patients who drink a lot of alcohol or have diabetes or rheumatoid diseases, and then use a lot of hormones. The causes and mechanisms of femoral head necrosis caused by hormones are still unclear. Most people believe that the use of hormones causes disorders of fat metabolism in the body, resulting in hyperlipidemia, which increases the number of fat cells in the femoral head and leads to microvascular embolism, thus causing ischemia and necrosis of bone cells in the femoral head. There is a time correlation between hormones and femoral head necrosis, and statistics show that the fastest onset can occur 3 months after the use of hormonal drugs, and the slowest onset can be as long as 5-10 years. Although some patients present with unilateral femoral head necrosis at the time of consultation, they may still present with bilateral femoral head necrosis as time progresses. Clinically, there is no special manifestation of hormonal osteonecrosis of the femoral head, which mostly manifests as hip pain, muscle atrophy, deep pressure pain in the groin area and lateral hip, etc.