Tension-adjusted T wave is a mechanism by which the heart adjusts the T wave to be in phase with the main wave of the QRS wave group in order to adapt to the abnormal intraventricular excitation sequence, which has the effects of tension adjustment, cardiac memory phenomenon and accumulation. ( 1) Tension adjustment effect: When the ventricular excitation sequence is changed, the T wave polarity is made to be in phase with the main wave of QRS wave group of abnormal depolarization through the tension adjustment effect of cardiac excitation, i.e., the ventricular depolarization changes adjust the ventricular repolarization sequence. ( 2) Cardiac memory phenomenon: When the heart resumes sinus rhythm after a period of altered excitation sequence, it still remembers the abnormal ventricular excitation sequence, and the ECG shows persistent T-wave inversion (the polarity is in phase with the main wave of the QRS wave group at the time of abnormal excitation sequence). (3) Cumulative effect: It means that the change in the excitation sequence must be repeated for a certain period of time and a certain number of times before the tensor-adjusted T-wave reaches its maximum effect, and the duration of the T-wave change can be divided into short-term and long-term memory. Short-term memory refers to the T-wave changes caused by the pacing time from 15 min to 2 h. These changes usually last from a few minutes to a few hours. Long-term memory is the T-wave change after 2 to 3 weeks of pacing, which can last from several weeks to several months. Common clinical manifestations: 1) T-wave changes after ventricular tachycardia (VT) 2) T-wave changes after right ventricular pacing 3) T-wave changes after ventricular pre-excitation 4) T-wave changes after bundle branch block