Meningiomas are second only to gliomas in terms of incidence and account for approximately % of all intracranial tumors. The cause of meningioma is still unclear. It is thought to be related to alterations in the internal environment and genetic variants, but not due to a single factor. Cranial trauma, radioactive irradiation, and viral infections that mutate cell chromosomes or increase the rate of cell division may be associated with the development of meningiomas. In recent years, molecular biology studies have confirmed that meningiomas most commonly lack a gene fragment on 22 pairs of chromosomes. Surgical resection is the most effective treatment for saddle node meningiomas. Small tumors less than 3 cm in diameter are easier to be fully resected and can be cured; large tumors larger than 5 cm in diameter are more difficult to be fully resected because of the close relationship with the optic pathway, pituitary gland, hypothalamus, cavernous sinus, internal carotid artery and its branches. In recent years, with the development of modern diagnostic equipment such as CT, MRI, electrophysiological detection and the improvement of microsurgical techniques as well as the application of ultrasonic suction and laser knife, the total resection rate of saddle node meningioma has gradually increased and the surgical results have been improving. Since the tumor receives double blood supply from the internal and external carotid arteries, the tumor location is deeper and more bleeding during surgery, attention should be paid to the protection of hypothalamus, optic nerve, optic cross and pituitary stalk, and complete resection should include the invaded dura mater and the adjacent skull, otherwise it is easy to recur.