Interventional treatment of cirrhosis, portal hypertension and hypersplenism

  Cirrhosis is a common digestive system disease with common causes such as viral infections, long-term alcohol consumption, drug-related liver damage and parasitic diseases, the first two of which are predominant in China. Patients often have already developed into liver function decompensation stage with corresponding complications such as portal hypertension and hypersplenism, etc. Clinically, they often present with upper gastrointestinal bleeding, vomiting blood and/or passing black stools, abdominal distension, gingival and nasal bleeding, etc. Signs are mainly varices in the abdominal wall, ascites, umbilical hernia, hard liver and enlarged spleen. The examination is mainly for esophagogastric fundic varices and decreased white blood cells, red blood cells, platelets, and liver function changes. The current conservative treatment is ineffective. Interventional minimally invasive flow dissection and shunt treatment with less damage and positive effect has gradually replaced other treatments and is accepted by more and more patients.  Percutaneous hepatic puncture portal variceal vein embolization The method is to puncture the portal vein of the liver through the skin and embolize the varicose vein by placing a catheter into the varicose vein through the portal vein, thus closing all varices and stopping bleeding or no risk of bleeding. The varices closed by this method are equivalent to the trunk of a tree, and the varices of the esophagogastric fundus are equivalent to the branches and leaves of a tree, and when the trunk dies, the branches and leaves disappear naturally. The indications are the same as the method.  Partial embolization of splenic artery is mainly applied to hypersplenism (splenomegaly, leukocyte and platelet reduction) caused by portal hypertension in cirrhosis. The reason is that in the above patients, blood cells are mainly destroyed in the spleen, and partial splenic embolization causes partial necrosis of the spleen, so that the destruction of blood cells in the spleen is reduced or disappears, which solves hypersplenism and preserves the normal immune function of the spleen at the same time.