Troponin is an important predictive tool for cardiovascular disease, especially for the diagnosis of acute coronary artery disease with a high degree of specificity and sensitivity. Elevated troponin generally refers to myocardial cell injury or necrosis, such as acute myocardial infarction and acute myocarditis. It is also elevated in acute left heart failure and rapid arrhythmias. In addition, non-ischemic myocardial damage can also cause troponin elevations, such as neurogenic myocardial injury. The metabolism of the central nervous system is closely related to the function of the heart. Diseases such as cerebrovascular stenosis may cause inadequate perfusion of blood in the posterior hypothalamus, which in turn may lead to a series of manifestations such as autonomic dysfunction, increased sympathetic excitability, increased release of catecholamines, and decreased myocardial contractility, ultimately causing an increase in troponin; other diseases such as abnormal renal function and pulmonary embolism may also cause an increase in troponin.