1. The patient had a sudden onset of epigastric pain on September 28, 2008 with no obvious cause, with persistent distension, accompanied by vomiting, the vomit is stomach contents, not accompanied by fever, diarrhea, jaundice and other symptoms, and was seen in our emergency room, blood biochemistry showed: blood lipase 830u/L, blood amylase 1300 u/L, blood glucose 9.08mmol/L, blood cell analysis showed: white blood cells 12.2×109/L, NEU% 88.5%; abdomen B ultrasound showed: large tail of pancreatic body with poor visualization, gallbladder stones and cholecystitis; CT of upper abdomen showed: pancreatitis, cholecystitis, and hypodense occupancy in the right lobe of liver. He was admitted to the general surgery department as “acute pancreatitis, gallbladder stones and cholecystitis”. On admission: body temperature 38℃, respiration 22 times/min, pulse 78 times/min, blood pressure 150/102mmHg, clear consciousness, no yellowish staining and fresh bleeding spots on the skin and mucous membranes, no superficial lymph node enlargement, a small amount of wet rales can be heard in both lungs, heart sounds are strong and rhythmic, abdomen is slightly dilated, no gastrointestinal pattern and peristaltic waves, abdominal muscles are slightly tense, no obvious pressure pain in the whole abdomen, drum sound on percussion. There was no significant pressure pain in the whole abdomen, drum sound on percussion, negative mobile turbid sounds, and weak intestinal sounds on auscultation. After one week of treatment, the patient’s condition was basically stable, and a repeat CT examination showed pancreatic pseudocyst formation. After 74 days of treatment, the patient’s vital signs were stable and he could eat normally by mouth, and the laboratory tests such as blood cell analysis and blood biochemistry were basically normal. After the patient’s condition was stabilized, a jejunal nutrition tube was placed on the 12th day of hospitalization. The following diagram shows that the position of the jejunal nutrition tube was clearly defined by taking a standing abdominal radiograph when the jejunal nutrition tube was placed: Enteral nutrition The drip rate was gradually increased from 20ml/hour to 58ml/hour, while the intravenous nutrition solution was reduced accordingly, so that the patient could gradually transition from parenteral nutrition to enteral nutrition, and on the 56th day of admission, the patient was allowed to eat a small amount of liquid food by mouth, and the amount of food was gradually increased, while the jejunal nutrition tube assisted the treatment. At the end of the treatment, the patient’s physiological needs were basically met by oral feeding. At the end of the treatment, the patient’s oral feeding could basically meet the physiological needs. The nutrition supply during the whole treatment is shown in the following figure: 3. The trends of the laboratory indicators were analyzed, and the results are as follows: (1) Two important indicators for the diagnosis of pancreatitis are blood and urine amylase and lipase, the patient was checked for urine amylase 11,367 U/L after admission, and the second reexamination rapidly decreased to 390 U/L and returned to normal, so no further testing was performed. (2) The overall trend of leukocytes in blood cell analysis was decreasing, indicating that the infection symptoms were gradually relieved, and the lymphocytes had a slightly increasing trend, initially the lymphocytes were below the normal value, and after a period of treatment, the lymphocytes gradually returned to normal, indicating that the immune function was enhanced, the results are shown in Figure 3: (3) The two main indicators of renal function were creatinine and (3) The two main indicators of renal function are creatinine and urea nitrogen. At the time of admission, creatinine and urea nitrogen were higher than normal, and after treatment, the overall trend was down, indicating that the patient’s renal function gradually improved. In addition, the patient’s fasting diet may be insufficient nitrogen supplementation, resulting in low protein and malnutrition, so he was given symptomatic treatment such as liver protection and human albumin infusion. 4. Imaging changes (1)2008-9-28: This is the abdominal CT at the time of admission, suggesting peripancreatic inflammatory exudate. (2)2008-10-13: This is the rechecked abdominal CT 14 days after admission, suggesting peripancreatic pseudocyst formation. (3) 2008-10-23: This is the review of abdominal CT at 24 days after admission, suggesting that the pseudocyst was absorbed and reduced.