Can genital herpes be cured? When you accidentally get genital herpes, you can find out from the information that this skin infection is not untreatable and cured, but it can be harmful if left untreated. Therefore, if you are infected with genital herpes, then we need to know more about it. The genital herpes occurs in the genital area of the herpes simplex, is a viral infectious skin disease, Chinese medicine belongs to the category of hot sores, it is different from the herpes simplex that occurs outside the corners of the mouth, the vast majority of the transmission through sexual relations. The disease is self-limiting and can heal itself in about 1-2 weeks. The goal of treatment is to prevent the next recurrence. The principle of treatment is to shorten the duration of the disease, prevent secondary infections and reduce recurrence. Genital herpes is not a disease of no; however, it is not treated in a timely manner. Herpes. Herpes can also be harmful! So be actively treated! Treatment must choose the regular hospital! Genital herpes conventional treatment methods Corrosive drugs, some national drugs are also corrosive and can only be used on the wart site, because there is corrosive can not be used on normal skin, so for subclinical infection and latent virus can not be removed, the treatment will recur. The actual antiviral drugs and immunity boosting drugs may temporarily keep the virus in a suppressed state, but without the drugs it will come back, and the immunity boosting drugs work on the blood and dermis, so the virus in the superficial skin mucosa cannot establish an effective immune response, and will come back after treatment. How is genital herpes caused Genital herpes is caused by about 85% to 90% of herpes simplex virus type 2 (HSV-2) and 10% to 15% of herpes simplex virus type 1 (HSV-1). HSV is a DNA virus with a core of linear double-stranded DNA with a molecular weight of 160×10Da and a stereosymmetric 20-sided protein capsid consisting of 162 capsid particles. The HSV genome encodes virus-specific core proteins and structural proteins, including glycoproteins present on the surface of the virus, which induce the production of neutralizing antibodies in the body. The HSV genome also encodes non-structural proteins, such as thymidine kinase (TK) and DNK polymerase, which are important for viral replication. Since these viral enzymes are different from cellular enzymes, some antiviral drugs that selectively inhibit viral enzymes, such as acyclovir, have been developed accordingly. Primary genital herpes is contracted through direct contact with a person infected with the herpes virus through sexual intercourse. HSV invades epithelial cells through minor abrasions or fissures in the skin mucosa, where it replicates and multiplies, causing inflammatory reactions such as ballooning degeneration and necrosis of cells, infiltration of inflammatory cells, and stimulation of the body’s immune response. The virus that invades the organism travels up the peripheral sensory nerves into the sensory or motor ganglia of the dorsal root of the crista medullaris, establishing a latent infection, which is the root cause of genital herpes recurrence. Activation of the latent virus is associated with viral thymidine kinase, etc. and immune and non-immune factors of the body. The latent virus is activated and travels down the peripheral nerve axons to the skin and mucous membranes, causing recurrence. What drugs to take for genital herpes I. Antiviral therapy Acyclovir, valacyclovir, and famciclovir for genital herpes can reduce symptoms, shorten the course of the disease, and reduce detoxification. (I) Initial infection
Acyclovir 200mg, 5 times/d for 7-10d; or acyclovir 400mg, 3 times/d for 7-10d; or valacyclovir 300mg, 2 times/d for 7-10d, or famciclovir 250mg, 3 times/d for 7-10d. (II) Recurrent infection
It is best to start treatment within 24 h of the onset of prodromal symptoms or damage. Acyclovir 200 mg, 5 times/d, for 5 d; or acyclovir 400 mg, 3 times/d, for 5 d; or valacyclovir 300 mg, 2 times/d, for 5 d; or famciclovir 125-250 mg, 3 times/d, for 5 d. (C) Patients with frequent relapses (more than 6 relapses in 1 year)
To reduce the number of relapses, suppressive therapy is available. Acyclovir 400mg, orally, 2 times/d; or valacyclovir 300mg, orally, 1 time/d; or famciclovir 125-250mg, orally, 2 times/d. All the above drugs need to be taken for a long time, usually for 4 months to 1 year. (iv) Severe infections Refer to those with severe symptoms of primary infection or extensive skin lesions. Acyclovir 5mg~10mg/kg body weight each time, intravenous drip, 1 time/8h, with 5~7d or until the clinical symptoms subside. (e) Genital herpes occurring in immunodeficient individuals can be treated according to the above protocol, and if necessary, the course of treatment can be extended appropriately. (vi) Herpes in pregnancy
The advantages and disadvantages of mother and child need to be weighed against the choice of appropriate antiviral drugs and regimens. It is currently advocated that pregnant women with initial genital herpes can be treated with oral acyclovir; those with severe complications that can be life-threatening should be treated with intravenous acyclovir; for pregnant women with frequent recurrences or new infections of genital herpes at full term, treatment with acyclovir can be used to reduce the appearance of active damage and thus reduce the rate of cesarean delivery; for pregnant women with a previous history of recurrent genital herpes but no recurrence at near full term In pregnant women with a previous history of recurrent genital herpes but no signs of recurrence in the last full term, acyclovir treatment may be withheld. (vii) Neonatal herpes Acyclovir 30-60 mg/(kg.d) intravenously for 10-21 days.