Hyperlipidemia has become a widespread and persistently elevated problem in our population, which means that it is necessary for many people to place lipid-modifying therapy on their agenda. The primary goal of lipid-modifying therapy is to lower LDL, and treatment strategies and control goals need to vary from person to person. For example, the Guidelines for the Prevention and Treatment of Dyslipidemia in Chinese Adults state that LDL should be controlled below 2.59 mmol/l for patients who already have coronary heart disease or diabetes, and below 2.07 mmol/l for patients with ischemic cardiovascular disease combined with diabetes. What is LDL? Blood lipids mainly contain triglycerides and cholesterol, which are mainly derived from the food we eat everyday and synthesized in our body. Cholesterol can be divided into LDL cholesterol and HDL cholesterol, LDL cholesterol is the “bad” cholesterol and HDL cholesterol is the “good” cholesterol. Dyslipidemia mainly refers to the increase of triglycerides and “bad” cholesterol, and low “good” cholesterol, which are the main causes of coronary heart disease and cerebral thrombosis. The increase of triglycerides and “bad” cholesterol can directly lead to damage of vascular endothelial function. For example, oxidation of “bad” cholesterol can induce damage to the endothelium, causing endothelial cell degeneration, necrosis and detachment, thus affecting the function of the endothelium. After endothelial damage, the subendothelial layer is exposed, and the increased “bad” cholesterol and other lipids in the blood will enter the vessel wall through the damaged endothelium and deposit in the subendothelium, thickening the endothelium, and at the same time, platelets can rapidly adhere and gather in the damaged area, gradually forming atherosclerotic plaques. Atherosclerotic plaques can be divided into stable plaques and unstable plaques. Stable plaques are not easy to rupture, but will gradually become larger, narrowing the lumen of blood vessels, resulting in reduced blood supply to the heart and brain, thus causing angina and cerebral blood supply deficiency; unstable plaques are prone to rupture, and the material gushing out from the plaques will form blood clots, blocking the heart and brain vessels and causing acute myocardial infarction, stroke and sudden death. The “good” cholesterol has a series of biological activities against atherosclerosis, mainly including promoting intracellular cholesterol outflow, anti-inflammatory and antioxidant effects, which have a direct protective effect on the arterial blood vessel wall and can promote the rapid regression of atheromatous plaques, if the level is too low, it is not conducive to the protection of vascular health. If the level is too low, it is not conducive to the protection of vascular health. High LDL can be regulated by diet and nutrition, usually eating less greasy and sweet food, more vegetables and fruits, and proper exercise. Absorb more vegetable protein and less animal protein. Second, the diet of people with high LDL 1, more fish (especially seafood), soybeans and soy products, poultry, lean meat and other foods that can provide high quality protein, while saturated fatty acids, cholesterol is low. 2.Control the intake of animal liver and other offal, and strictly limit the intake of animal brain, crab meat, fish roe, etc. 3.Cook with vegetable oil and minimize the intake of animal fats. 4.Eat more vegetables, fruits, coarse grains, etc., to ensure the right amount of food fiber, vitamins, inorganic salt intake.