Arterial entrapment is the gradual peeling and expansion of the intima due to a localized tear in the intima, which is subjected to a strong blood shock, creating two lumens, true and false, within the artery. This leads to a number of manifestations including tear-like pain. The greatest danger of aortic coarctation is death. The aorta is the main blood vessel of the body, subjected to pressure directly from the beating heart, with tremendous blood flow. The occurrence of a tear in the intimal layer has a very high chance of rupture and a very high mortality rate if proper and timely treatment is not provided. Previous literature reported mortality rates of up to 50% within 1 week and between 60-70% within a month. In addition to this, even if the patient survives, the enlargement of the false lumen and the increase in pressure, which reduces the blood flow to the true lumen vessels, leads to ischemia of the organs in the area supplied by the aorta. The main treatment options for aortic coarctation include conservative, interventional and surgical treatment. Interventional endoluminal repair techniques have enriched the treatment of aortic coarctation and have made the procedure less invasive and safer. Conservative treatment For patients with acute coarctation, regardless of the further treatment options, the first step is to provide the appropriate conservative treatment: blood pressure control and pain control. This usually requires the application of powerful drugs such as sodium nitroprusside for hypotension and morphine for analgesia. For patients in critical condition, emergency tracheal intubation, ventilator-assisted breathing, and emergency resuscitation surgery are often required, but this also implies a very high risk and mortality rate. Surgical and interventional treatment After the patient’s condition has been appropriately stabilized, the choice of treatment depends mainly on the type of entrapment. For the current state of treatment, for Stanford type B aortic coarctation, minimally invasive endoluminal treatment is the mainstay. The basis for treatment includes the following conditions, or indications for surgery: persistent enlargement of the entrapment, as evidenced by a rapidly increasing diameter and extent of the aortic entrapment, thoracic hemorrhage, and uncontrollable pain; or ischemia of major branches of the aorta, such as the superior mesenteric artery and renal artery. Traditional minimally invasive endoluminal repair of aortic coarctation technically requires at least a 1.5 cm anchorage zone on the aorta to prevent incomplete proximal occlusion and endoleaks. However, with improvements in endoluminal repair devices and advances in endoluminal repair techniques, this indication has been expanded to allow treatment of Stanford type B aortic coarctation with the main fissure within 1.5 cm of the left subclavian artery opening by hybridization or various endoluminal repair coarctures (chimney, open window, modular branch stent) For Stanford type A aortic coarctation with the fissure in the ascending aorta endoluminal The repair has been suggested by placing an overlapping stent in the ascending aorta to isolate the proximal clamping fissure, but this procedure requires specific anatomic constraints. In the acute phase, ascending aortic replacement is performed, and Sun’s procedure remains the primary treatment for type A aortic coarctation today.