Hypotension after cerebral infarction may be due to autonomic dysfunction caused by damage to the corresponding brain functional areas.
Cerebral infarction is a critical condition in neurology and is the most common cerebrovascular disease, which is characterized by high morbidity, disability and mortality.
Its clinical manifestations are related to the location, size and number of infarcts. It can be manifested as one or more of motor, sensory, consciousness, emotion, cognition, higher cortical function and autonomic function.
If the patient’s injury is to the thalamus or corresponding fiber tracts, it will cause autonomic dysfunction, resulting in sympathetic excitation manifesting as excessive sweating, hypertension, increased heart rate, urinary and fecal incontinence, etc. or parasympathetic excitation manifesting as low blood pressure, slow heart rate, lethargy, etc. The patient’s vital signs should be closely monitored, and the patient should have a good understanding of the symptoms.
The patient’s vital signs should be closely monitored, and the patient should actively cooperate with the professional physician’s treatment.