There are three main factors that contribute to the development of lumbar disc herniation: aging, trauma, and lumbar muscle strain. With age, the main components of the intervertebral disc, i.e., the annulus fibrosus and the nucleus pulposus, gradually decrease in water content, the intervertebral disc becomes thinner, and the nucleus pulposus gradually loses its elasticity, resulting in the loosening of the intervertebral disc structure. At this time, under the action of some stress (such as lumbar sprain), the nucleus pulposus pushes up the annulus fibrosus, causing the disc to “bulge” or “protrude”. If the disc is damaged or even ruptured, the annulus fibrosus becomes weak. In mild cases, the nucleus pulposus can “protrude”; in severe cases, the nucleus pulposus can be pushed out along the broken area of the annulus fibrosus, which is called “prolapse”. Whether “herniated” or “prolapsed”, the disc may compress the nerves traveling through the spinal canal and cause the clinical symptoms described above. The occurrence of lumbar disc herniation is certainly related to factors such as aging and lumbar activity. However, its triggers are not only lumbar spine trauma or lumbar weight bearing. Because clinically, the vast majority of patients do not have a history of obvious injuries, and many patients are even brain workers. In fact, chronic strain on the low back and lack of muscle strength around the lumbar spine are the most important causes of lumbar disc herniation.