The statement “90% recovery from Hunter’s syndrome” is not scientific. Clinically Hunter’s syndrome is generally poorly treated and often has sequelae. Most patients cannot recover through conservative treatment and need early surgical treatment. Hunter’s syndrome is also known as geniculate ganglionitis. After the geniculate ganglion is infected by varicella zoster virus, the virus lurks in the geniculate ganglion of the facial nerve. When the body’s resistance drops, varicella zoster virus will come out to replicate and activate, and the nerve is infected by the virus and undergoes inflammation, denaturation, edema, and geniculate ganglion lesions appear. It manifests as peripheral facial nerve paralysis with severe pain behind the ear, dulled sensation in the auricle or external auditory canal, herpes of the tympanic membrane and external auditory canal, and may be accompanied by taste disturbance in the anterior 2/3 of the tongue and impaired secretion of the lacrimal and salivary glands. It can be treated with antiviral therapy, glucocorticoids, or surgery, but it is also more difficult to repair damaged nerve tissue, and residual neuralgia can easily occur. If not treated promptly, this pain lasts for months or longer. If Hunter’s syndrome is suspected, it is recommended to visit a hospital ear, nose and throat department or neurology department for early and prompt treatment under the guidance of a physician.